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Motohiko Kadoki on Muck Rack

Motohiko Kadoki

As seen in: Nature, PNAS, Cell Press, The Journal of Immunology
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Articles

A missense variant in SLC39A8 confers risk for Crohn’s disease by disrupting manganese homeostasis and intestinal barrier integrity

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By Toru Nakata, Elizabeth Creasey, Motohiko Kadoki, Helen Lin
| PNAS Verified
A missense variant in SLC39A8 confers risk for Crohn’s disease by disrupting manganese homeostasis and intestinal barrier integrity Toru Nakata, Elizabeth A. Creasey, Motohiko Kadoki, Helen Lin, Martin K. Selig, Junmei Yao, Ariel Lefkovith, Mark J. Daly, Daniel B. Graham, Ramnik J. Xavier Proceedings of the National Academy of Sciences Nov 2020, 202014742; DOI: 10.1073/pnas.2014742117
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Cell Type- and Stimulation-Dependent Transcriptional Programs Regulated by Atg16L1 and Its Crohn's Disease Risk Variant T300A

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By Mukund Varma, Motohiko Kadoki, Ariel Lefkovith, Kara L. Conway Verified
| The Journal of Immunology Verified
Mukund Varma, Motohiko Kadoki, Ariel Lefkovith, Kara L. Conway, Kevin Gao, Vishnu Mohanan, Betsabeh Khoramian Tusi, Daniel B. Graham, Isabel J. Latorre, Andrew C. Tolonen, Bernard Khor, Aylwin Ng and Ramnik J. Xavier J Immunol July 15, 2020, 205 (2) 414-424; DOI: https://doi.org/10.4049/jimmunol.1900750 Key Points Stimulation-based profiling defined genotype- and cell-specific Atg16L1 programs. Cell type and stimulation strongly influence Atg16L1 T300A genotypic differences.
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Cell Type- and Stimulation-Dependent Transcriptional Programs Regulated by Atg16L1 and Its Crohn's Disease Risk Variant T300A

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By Mukund Varma, Motohiko Kadoki, Ariel Lefkovith, Kara L. Conway Verified
| The Journal of Immunology Verified
Mukund Varma, Motohiko Kadoki, Ariel Lefkovith, Kara L. Conway, Kevin Gao, Vishnu Mohanan, Betsabeh Khoramian Tusi, Daniel B. Graham, Isabel J. Latorre, Andrew C. Tolonen, Bernard Khor, Aylwin Ng and Ramnik J. Xavier J Immunol June 10, 2020, ji1900750; DOI: https://doi.org/10.4049/jimmunol.1900750 Key Points Stimulation-based profiling defined genotype- and cell-specific Atg16L1 programs. Cell type and stimulation strongly influence Atg16L1 T300A genotypic differences.
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