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Pamela Wong on Muck Rack

Pamela Wong

  • Contributing Writer, Art Hag
  • Marketing Associate, Brooklyn Conservatory of Music
New York
As seen in: Nature, Wiley Online Library, BioRxiv, Cell Reports, The Journal of Clinical Investigation, Chemistry World, Art Asia Pacific, The Stony Brook Press
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Articles

Stroma-derived Dickkopf-1 contributes to the suppression of NK cell cytotoxicity in breast cancer - Nature Communications

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By Seunghyun Lee, Jennifer Tran, Qihao Ren, Julia Wang, Pamela Wong, Sheila Stewart, Cynthia X. MA, Todd A. Fehniger
| Nature Verified
Abstract Mechanisms related to tumor evasion from NK cell-mediated immune surveillance remain enigmatic. Dickkopf-1 (DKK1) is a Wnt/β-catenin inhibitor, whose levels correlate with breast cancer progression. We find DKK1 to be expressed by tumor cells and cancer-associated fibroblasts (CAFs) in patient samples and orthotopic breast tumors, and in bone.
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MicroRNA‐146a deficiency enhances host protection against murine cytomegalovirus

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By Pamela Wong, Jeffrey W Leong, Hyogon Sohn, Lily Chang
| Wiley Online Library Verified
Conflict of interest The authors declare no financial or commercial conflict of interest. Supporting Information Filename Description eji5848-sup-0001-FigureS1-S6.pdf476.7 KB Supporting Information References 1, and , The dynamic life of natural killer cells. Annu. Rev. Immunol. 2004; 22: 405–429. 2, Human natural killer cells. Blood. 2008; 112: 461–469. 3, , , and , Natural killer cell depletion enhances virus synthesis and virus-induced hepatitis in vivo. J. Immunol. 1983; 131: 1531–1538.
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Expression of a single inhibitory Ly49 receptor is sufficient to license NK cells for effector functions

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By Sytse J Piersma, Shasha Li, Pamela Wong, Michael Bern
| BioRxiv
Abstract Natural killer (NK) cells recognize target cells through germline-encoded activation and inhibitory receptors enabling effective immunity against viruses and cancer. The Ly49 receptor family in the mouse and killer immunoglobin-like receptor family in humans play a central role in NK cell immunity through recognition of MHC class I and related molecules. Functionally, these receptor families are involved in licensing and rejection of MHC-I-deficient cells through missing-self.
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