Skip To Main Content
Pernille Bülow on Muck Rack

Pernille Bülow

As seen in: BioRxiv, Cell Press
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with Pernille

Contact Pernille, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

GABAergic synaptic scaling is triggered by changes in spiking activity rather than AMPA receptor activation

|
By Carlos Gonzalez-Islas, Zahraa Sabra, Ming-fai Fong, Pernille Bülow
| BioRxiv
Abstract Homeostatic plasticity represents a set of mechanisms that are thought to recover some aspect of neural function. One such mechanism called AMPAergic scaling was thought to be a likely candidate to homeostatically control spiking activity. However, recent findings have forced us to reconsider this idea as several studies suggest AMPAergic scaling is not directly triggered by changes in spiking.
Open in Who Shared Issue with article?

GABAergic synaptic scaling is triggered by changes in spiking activity rather than transmitter receptor activation.

|
By Carlos Gonzalez-Islas, Zahraa Sabra, Ming-fai Fong, Pernille Bülow
| BioRxiv
Thank you for your interest in spreading the word about bioRxiv. NOTE: Your email address is requested solely to identify you as the sender of this article. Your Email * Your Name * Send To * Enter multiple addresses on separate lines or separate them with commas. Message Subject (Your Name) has forwarded a page to you from bioRxiv Message Body (Your Name) thought you would like to see this page from the bioRxiv website.
Open in Who Shared View Duplicates (692) Issue with article?

Homeostatic Intrinsic Plasticity Is Functionally Altered in Fmr1 KO Cortical Neurons

|
By T.J. Murphy, Gary J. Bassell, Pernille Bülow, Peter Wenner
| Cell Press Verified
Highlights • Activity block normally drives conversion of single- to multi-spiking neurons • Fmr1 knockout (KO) neurons fail to convert single- to multi-spiking neurons • Multi-spiking Fmr1 KO neurons display exaggerated compensatory increase in spiking • Altered homeostatic intrinsic plasticity may contribute to hyperexcitability in FXS Summary Cortical hyperexcitability is a hallmark of fragile X syndrome (FXS).
Open in Who Shared Issue with article?
See all 3 articles