Robert Tighe on Muck Rack

Robert Tighe

Auckland
As seen in: Environmental Health Perspectives, Nature, Wiley Online Library, Stuff.co.nz, Substack, Royal Society of Chemistry, The Journal of Allergy & Clinical Immunology, Respiratory Medicine
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Articles

Author Correction: Profibrotic monocyte-derived alveolar macrophages are expanded in patients with persistent respiratory symptoms and radiographic abnormalities after COVID-19 | Nature Immunology

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By Connor H. Puritz, Karolina J. Senkow, Nikolay S. Markov, Estefani Diaz, Emmy Jonasson, Rogan A. Grant, Hiam Abdala-Valencia, Robert Tighe, Ali Shilatifard, Jacob I. Sznajder, Karen M. Ridge, Alexander V. Misharin
| Nature Verified
Correction to: Nature Immunology https://doi.org/10.1038/s41590-024-01975-x, published online 4 October 2024. In the version of this article initially published, there was a typographical error in the first paragraph of the Methods, where in the sentence now reading “Subsequent CT scans were obtained in 29 patients a median of 118 d (range 31–249 d) after the first CT scan,” the median was first reported as “11 d.” The error has been corrected in the HTML and PDF versions of the article.
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Hyperpolarized 129Xe MRI and Spectroscopy of Gas Exchange Abnormalities in Chronic Hypersensitivity Pneumonitis

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By Madison Wulfeck, David G. Mummy, Shuo Zhang, Diana Kadi, Stacy M. Goins, Aparna Swaminathan, Robert Tighe, Bastiaan Driehuys, Joseph G. Mammarappallil
| Respiratory Medicine Verified
Keywords interstitial lung disease 129Xe MRI chronic hypersensitivity pneumonitis high-resolution computed tomography INTRODUCTION The spectrum of fibrotic interstitial lung diseases (ILDs) is characterized by inflammation and scarring of the lung parenchyma, which can lead to end-stage fibrosis. The significant heterogeneity in ILDs presents challenges in differentiating chronic hypersensitivity pneumonitis (CHP) from other fibrotic ILDs, specifically idiopathic pulmonary fibrosis (IPF) [1,2].
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Profibrotic monocyte-derived alveolar macrophages are expanded in patients with persistent respiratory symptoms and radiographic abnormalities after COVID-19 | Nature Immunology

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By Connor H. Puritz, Karolina J. Senkow, Nikolay S. Markov, Estefani Diaz, Emmy Jonasson, Rogan A. Grant, Hiam Abdala-Valencia, Robert Tighe, Ali Shilatifard, Jacob I. Sznajder, Karen M. Ridge, Alexander V. Misharin
| Nature Verified
Abstract Monocyte-derived alveolar macrophages drive lung injury and fibrosis in murine models and are associated with pulmonary fibrosis in humans. Monocyte-derived alveolar macrophages have been suggested to develop a phenotype that promotes lung repair as injury resolves.
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