Romain Barrès
France
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Male obesity causes adipose mitochondrial dysfunction in F1 mouse progeny via a let-7-DICER axis - Nature Communications
Abstract Male obesity negative affects gametic function and offspring metabolism. We here describe that (F0) obesity and weight loss in male mice reversibly alter metabolism and impair adipose mitochondrial function. These metabolic aberrations are transmitted to male offsprings (F1), which display reduced mitochondrial gene expression.
On the mechanisms of ultra-processed diet effects
Get full text access Log in, subscribe or purchase for full access. References 1. Astrup, A. ∙ Monteiro, C.A. Does the concept of “ultra-processed foods” help inform dietary guidelines, beyond conventional classification systems? Debate consensus Am. J. Clin. Nutr. 2022; 116:1489-1491 2. Astrup, A. ∙ Monteiro, C.A. Does the concept of “ultra-processed foods” help inform dietary guidelines, beyond conventional classification systems? NO Am. J. Clin. Nutr. 2022; 116:1482-1488 3. Monteiro, C.A. ∙ Astrup, A.
Effect of ultra-processed food consumption on male reproductive and metabolic health
Keywords ultra-processed diet reproduction hormones sperm quality diet intervention pollutants phthalates body weight management cholesterol depression score Introduction The consumption of ultra-processed foods (UPFs) has markedly increased globally,1 now accounting for over 50% of total energy intake in the UK,2 Australia,3 Canada,4 and the USA.5,6 UPFs are NOVA category 4 foods and defined as foods that are industrially processed and composed of highly transformed, derived, or synthesized...
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