Sarah Childs on Muck Rack

Sarah Childs

Kitchener
As seen in: Nature, Wiley Online Library, Cambridge University Press & Assessment, Memory Studies, PLOS Biology, Temple University News, openDemocracy, King's College London, eLife, New Bern Sun Journal and
Business Transformation Manager SSRP @yorkuniversity CIRPA Board. @thecarpentries instructor. ORCID 0000-0002-4450-4281 He/him
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Articles

What fish can reveal about brain blood vessels

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By Sarah Childs
| The Naked Scientists Podcast - Stripping Down Science
A gene called FoxF2 has been linked to cerebral vascular problems, including strokes, although quite how it's involved in the disease process wasn't known. So what Sarah Childs and Faye Graff, at the University of Calgary, have done to try to learn more was to use gene editing to deplete the number of working copies of the gene in zebrafish.
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A roadmap for establishing institutional readiness for cellular therapies targeting solid tumours and autoimmune diseases

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By Sarah Childs, David Ma, Elizabeth Houghton, Rasha Cosman, Jennifer Massey, Venessa T. Chin, Amanda L. McLaughlin, Simon Goodwin, Barbara Sheehan Withers, Callie Noakes, Annabel Horne, Jessica Boyd, Dion Forstner, Anthony J Rodrigues, Geeta Sandhu, Karim Ibrahim, Chloe Martin, Benjamin Kwan, Hergen Buscher, Philip J. Cunningham, Anthony M. Joshua, Laila Girgis, Ben Tran, Jayesh Desai, Simon Harrison, John Moore, Jia Liu, Valerie Valencia, Aaron O’Grady, Simone Kay Jensen, Helen Tao, Paul Preisz, Nicolas De Luca
| Nature Verified
Abstract Adoptive cellular therapies have revolutionised treatment of haematological malignancies and demonstrate potential in solid tumours and autoimmune disease. Real-world implementation remains limited by biological, logistical, and manufacturing barriers, restricting global uptake despite a rapidly expanding clinical trial portfolio. Effective implementation requires national and institutional readiness, multidisciplinary collaboration, scalable manufacturing, and robust infrastructure.
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Progressive mural cell deficiencies across the lifespan in a foxf2 model of cerebral small vessel disease

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By Emma EM Heeg, David Elliott, Sarah Childs
| eLife
eLife Assessment This study provides important insights into mural cell dynamics and vascular pathology using a zebrafish model of cerebral small vessel disease. The authors present convincing evidence that partial loss of foxf2 function results in progressive, cell-autonomous defects in pericytes accompanied by endothelial abnormalities across the lifespan.
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