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Satdarshan P Monga on Muck Rack

Satdarshan P Monga

Pittsburgh
As seen in: Nature, MDPI, The Conversation, Chron, Pittsburgh Post-Gazette, St. Louis Post-Dispatch, Cell Press, Houston Chronicle, ScienceAlert, Seattle PI and
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Articles

Hepatic adaptation to chronic metabolic stress primes tumorigenesis

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By Constantine N. Tzouanas, Jessica Shay, Marc Sherman, Benjamin E. Mead, Tyler T Dao, Junyan Tao, Brandon M Lehrich, George Eng, Jeffrey Patterson-Fortin, Titus Butzlaff, Miyeko Mana, Kellie E. Kolb, Chad Walesky, Brian Pepe-Mooney, Colton Smith, Sanjay Prakadan, Michelle L. Ramseier, Yuzhou Evelyn Tong, Julia Joung, Fangtao Chi, Woo-Jeong Jeong, Katherine J. Aney, Ethan Chen, Sahar Nissim, Feng Zhang, Vikram Deshpande Verified, Satdarshan P Monga, Georg M. Lauer, Wolfram Goessling, Omer Yilmaz, Ömer H. Yilmaz, Alex K. Shalek, Thomas McMahon-Skates, Carolyn L. Winston, Adam Rubin
| Cell Press Verified
Keywords single-cell genomics liver metabolism chronic stress response tissue memory epigenetic priming genetic perturbation computational methods development Introduction During chronic stress, cells must buffer environmental perturbations to ensure their own survival while contributing to tissue functions necessary for organismal health.1 Hepatocytes perform wide-ranging functions, including nutrient metabolism, protein secretion, and chemical detoxification.2,3 Moreover, they possess...
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Author Correction: Matrix viscoelasticity promotes liver cancer progression in the pre-cirrhotic liver - Nature

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By Foysal Rabbi, Aatur D Singhi, Louis Hodgson, Gregory W Charville, Satdarshan P Monga, Rebecca Wells, Ovijit Chaudhuri, Lóránd Váncza
| Nature Verified
Correction to: Nature https://doi.org/10.1038/s41586-023-06991-9 Published online 31 January 2024 In the version of this article initially published, due to mistakes when outputting final source data, the plot shown in Fig. 1n and Fig. 1n source data were incorrect. The conclusions are unaffected by the change. The source data and figure panel are updated in the HTML and PDF versions of the article.
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WNT–β-catenin signalling in hepatocellular carcinoma: from bench to clinical trials - Nature Reviews Gastroenterology & Hepatology

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By Satdarshan P Monga
| Nature Verified
Abstract WNT–β-catenin activation is observed in around 50% of all patients with hepatocellular carcinoma (HCC), through either gain-of-function mutations in CTNNB1 (which encodes β-catenin) or loss-of-function mutations in AXIN1 or APC. Currently, first-line therapies for HCC are immune checkpoint inhibitor (ICI) combinations, and β-catenin-active HCCs have garnered increased attention due to their unique tumour immune microenvironment (TIME).
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