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Main text (Immunity 25, 237–247; August 2006) Image misplacements were identified in Figure 5. Specifically, in Figure 5A bottom (Th2) and Figure 5B, the left and right images were inadvertently placed in the wrong order during figure assembly. The corrected figure now shows the correct order. This correction does not affect the conclusions of the paper.
1 Introduction Systemic lupus erythematosus (SLE) is an autoimmune disease involving aberrant immune responses, production of autoantibodies, and multiorgan system involvement. The etiology of SLE is multifactorial, likely involving a complex interplay between genetic risk factors and environmental triggers (1). Dysregulation of several innate and adaptive immune signaling pathways contributes to the inflammation characteristic of the disease.
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