Scott Armstrong on Muck Rack

Scott Armstrong

Dubai
As seen in: Mentl.Space, The New York Times, Wiley Online Library, Substack, Cell Press, New York Review of Books, Arabian Business (Arabic), Arabian Business (English), Robb Report, PR Week, Winnipeg Free Press and
Founder @mentl_space | Building healthier workplace cultures through evidence, stories and conversation | Together we thrive | 101,000+ subscribers on YouTube.
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Articles

Genetic variation reveals a homeotic long noncoding RNA that modulates human hematopoietic stem cells

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By Peng lyu, Gaurav Agarwal, Chun-Jie Guo, Adam Sychla, Wallace Bourgeois, Tianyi Ye, Chen Weng, Mateusz Antoszewski, Samantha Joubran, Alexis Caulier, Michael Poeschla, Scott Armstrong, Silvi Rouskin, Vijay Sankaran
| Cell Press Verified
Keywords genetic variation lncRNA HOXA9 SRSF2 splicing hematopoietic stem cell hematopoiesis leukemia clonal hematopoiesis Introduction The clustered homeobox (HOX) genes encode highly conserved transcription factors that are essential for embryonic development, particularly in establishing axial body patterning and defining segmental identity.1,2,3,4 Among these, the HOXA locus, first discovered as regulators of body segmentation, has more recently been identified as critical for normal and...
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Epigenetic Therapies

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By Wallace Bourgeois, Scott Armstrong, Emily B. Heikamp
| Cold Spring Harbor Perspectives in Medicine (Online)
Division of Hematology/Oncology, Boston Children's Hospital, Department of Pediatric Oncology, Dana-Farber Cancer Institute, and Harvard Medical School, Boston, Massachusetts 02215, USA Correspondence: emilyb_heikamp{at}dfci.harvard.edu
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Targeting the Menin-KMT2A interaction in leukemia: Lessons learned and future directions

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By Florian Perner, Jayant Y. Gadrey, Scott Armstrong
| Wiley Online Library Verified
1 ONCOGENIC KMT2A-COMPLEXES IN LEUKEMIA In mammalian cells six members of the Mixed-Lineage Leukemia (MLL)-family (MLL1/KMT2A, MLL2/KMT2B, MLL3/KMT2C, MLL4/KMT2D, SETD1A/KMT2F and SETD1B/KMT2G) encode for methyltransferases that catalyze Histone 3 lysine 4 mono-, di- and tri-methylation (H3K4me1/2/3) at promotors and enhancer regions and influence transcription.
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