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Shaela Fields

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Articles

Rbm5 sustains leukemia stem cells through a Myc-driven transcriptional circuitry

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By Mengli Zhang, Qiong Zhang, Yuxin Ouyang, Ye Xin, Jianxiang Zhang, Hong liu, Jianfeng Shen, Beisi Xu, Jia Chen, Depei Wu, Chunliang Li, Peng Xu, Shaela Fields
| Nature Verified
Abstract Acute myeloid leukemia (AML) represents a type of malignant hematological disease that is usually caused by the dysregulated developmental program of leukemia stem cells (LSCs). Here, we report that an unappreciated RNA-binding protein, Rbm5, selectively promotes murine leukemogenesis, maintains LSC self-renewal in vivo, and is dispensable for normal hematopoiesis.
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