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Shuying Mao

As seen in: BioRxiv, Cell Press
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Articles

Astrocyte Ca2+ activity regulates node of Ranvier length in the white matter

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By Jonathan Lezmy, Tania Quintela-López, Nicholas Woermann, Shuying Mao
| BioRxiv
Abstract Nodes of Ranvier generate action potentials along myelinated axons, but it is unclear whether they can modulate neural circuit function. Computer modelling previously predicted that adjusting node length can control axonal conduction speed1, but it is unknown whether mechanisms controlling nodal structure operate in the healthy brain.
Open in Who Shared Issue with article?

Parental origin of transgene modulates amyloid-β plaque burden in the 5xFAD mouse model of Alzheimer’s disease

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By Andrew Octavian Sasmita, Erinne Ong, Taisiia Nazarenko, Shuying Mao, Lina Komarek, Maik Thalmann, Veronika Hantáková, Lena Spieth, Stefan A. Berghoff, Helena J. Barr, Maximilian Hingerl, Friederike Börensen, Johannes Hirrlinger, Mikael Simons, Beth Stevens, Constanze Depp, Klaus-Armin Nave
| Cell Press Verified
Keywords 5xFAD Alzheimer’s disease amyloid-β light-sheet microscopy amyloid plaque transgenic mouse Thy1 promoter Introduction Published almost 20 years ago, the 5xFAD mouse model serves as a robust and widely used tool to study amyloidosis in Alzheimer’s disease (AD) in vivo.
Open in Who Shared View Duplicates (2) Issue with article?

Astrocyte Ca2+ activity regulates node of Ranvier length in the white matter

|
By Jonathan Lezmy, Tania Quintela-López, Nicholas Woermann, Shuying Mao
| BioRxiv
Abstract Nodes of Ranvier generate action potentials along myelinated axons, but it is unclear whether they can modulate neural circuit function. Computer modelling previously predicted that adjusting node length can control axonal conduction speed1, but it is unknown whether mechanisms controlling nodal structure operate in the healthy brain.
Open in Who Shared Issue with article?
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