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Svante Pääbo

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Articles

Genetic diversity of late Neanderthals in northwestern Europe

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By Elena Essel, Stéphane Peyrégne, Arev P. Sümer, Leonardo N. M. Iasi, Christian Heide, Divyaratan Popli, Cesare de Filippo, Marie-Theres Gansauge, Lars Gerullat, Laurin Lippik, Sarah Nagel, Birgit Nickel, Barbara Schellbach, Anna Schmidt, Johann Visagie, Antje Weihmann, Hugo Zeberg, Julia Zorn, Hélène Rougier, Isabelle Crevecoeur, Patrick Semal, Gregory Abrams, Thibaut Deviese, Stéphane Pirson, Kévin Di Modica, Pierre Cattelain, Christelle Draily, Michel Toussaint, Isabelle De Groote, Frido Welker, Cosimo Posth, Marie Soressi, Johannes Krause, Svante Pääbo, Matthias Meyer, Janet Kelso, Benjamin M Peter, Mateja Hajdinjak
| Nature Verified
Abstract Archaeological, osteological and genetic evidence suggests that Neanderthals lived in small groups1,2; however, less is known about whether these groups were part of isolated communities or belonged to larger, well-connected populations3. The dense concentration of broadly contemporaneous Neanderthal sites in the Meuse Basin, Belgium4, provides a rare opportunity to study regional populations at high resolution.
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Regulation of NAT1 activity in modern humans by a novel phosphorylation site

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By Youngmin Ko, John Hintze, Luise Fast, Mikel Lana Alberro, Eva Rakava, Richard Ågren, Stephan Riesenberg, Wieland B. Huttner, Janet Kelso, Svante Pääbo, Hugo Zeberg
| Science Magazine Verified
Abstract INTRODUCTION RESULTS DISCUSSION MATERIALS AND METHODS Acknowledgments Supplementary Materials REFERENCES Information & Authors Metrics & Citations View Options References Figures Tables Media Share Abstract N-acetyltransferase 1 (NAT1) is an enzyme that acetylates certain drugs and carcinogens and is involved in folate metabolism. Some NAT1 variants are associated with susceptibility to cancer and birth defects.
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Search-and-remove genome editing allows selection of cells by DNA sequence - Nature Communications

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By Philipp Kanis, Theresa Schaffer, Svante Pääbo, Luise Fast, Madina Omar
| Nature Verified
Abstract The selection of cells that have acquired a desired gene edit is often done by the introduction of additional genes that confer drug resistance or encode fluorophores. However, such marker genes can have unintended physiological effects and are not compatible with editing of single nucleotides. Here, we present SNIPE, a method that allows the marker-free selection of edited cells based on single nucleotide differences to unedited cells.
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