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T. Alex Perkins on Muck Rack

T. Alex Perkins

As seen in: British Journal of Cancer, Nature, PLOS, PNAS
Professor of Biological Sciences at the University of Notre Dame. Mathematical modeling of infectious disease dynamics and control. Views my own.
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Articles

Decomposing Influenza-Like Illness: Estimating Pathogenic Drivers of Syndromic Indicators

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By Kelsey Shaw, T. Alex Perkins
| preprints.org
You are already at the latest version Syndromic indicators of respiratory disease such as influenza-like illness (ILI) are commonly monitored by public health agencies and used in decision making. The diagnostic criteria for these indicators are based on clinical symptoms, not pathogen detection, and thus are a useful view into the overall burden of respiratory disease resulting from a myriad of pathogens.
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Implications of successive blood feeding on Wolbachia-mediated dengue virus inhibition in Aedes aegypti mosquitoes - Nature Communications

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By Rebecca Johnson, Mallery I. Breban, Isabel M. Ott, Perran A. Ross, Xinyue Gu, Nathan D. Grubaugh, T. Alex Perkins, Doug E. Brackney
| Nature Verified
Abstract Wolbachia is a promising strategy to inhibit dengue virus (DENV) transmission by Ae. aegypti mosquitoes. Laboratory studies assessing DENV inhibition by Wolbachia typically have not considered natural frequent mosquito blood feeding behavior. Here, we determine the impact of successive feeding on DENV-2 transmission by Ae. aegypti in the presence or absence of Wolbachia (wAlbB and wMelM strains).
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Human movement and environmental barriers shape the emergence of dengue - Nature Communications

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By Vinyas Harish, Felipe J. Colón-González, Megan Davis, David M Pigott, T. Alex Perkins, DANIEL WEISS, Gonzalo M. Vazquez-Prokopec, Gerson Barbosa, Nuno R Faria, Simon I Hay, Fabián Correa-Morales, Francisco Chiaravalloti-Neto
| Nature Verified
Abstract Understanding how emerging infectious diseases spread within and between countries is essential to contain future pandemics. Spread to new areas requires connectivity between one or more sources and a suitable local environment, but how these two factors interact at different stages of disease emergence remains largely unknown. Further, no analytical framework exists to examine their roles.
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