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Tianying Zhang on Muck Rack

Tianying Zhang

As seen in: Nature, Wiley Online Library, New Phytologist, Cell Press, Cell Reports, Future Medicine
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Articles

Krüppel-like factor 2 programs early exhausted T cell states and restrains antiviral immunity

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By Zifeng Li, Wenhao Li, Huiqing Liang, Fan Xu, Leilei Zhang, Wei Zhang, Yanhan Wang, Yurui Zhou, Shijie Luo, Xing Lei, Hailang Li, Wei-Feng Huang, Meixia Che, Wen-Hsien Liu, Jialiang Huang, Tianying Zhang, Kejia Wang, Nengming Xiao, Kairui Mao, Yajun Jiang, Hongling Huang, Shengjun Geng, Shengyong Tao
| Cell Press Verified
Keywords CD8+ T cell chronic infection in vivo pooled CRISPR screening CX3CR1 KLF2 S1PR1 PD-1 immunotherapy TNF anti-viral effector-like cells T cell exhaustion immunopathology Get full text access Log in, subscribe or purchase for full access. References 1. Omilusik, K.D. ∙ Goldrath, A.W. Remembering to remember: T cell memory maintenance and plasticity Curr. Opin. Immunol. 2019; 58:89-97 2. Herndler-Brandstetter, D. ∙ Ishigame, H. ∙ Shinnakasu, R. ...
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Short-chain acyl-CoA dehydrogenase initiates mtDNA demethylation and leakage to fuel antitumor immunity in colorectal cancer

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By Fang Yang, Meng Wang, Shaofan Hu, Xu Guan, Kun Zhao, Yong Zhou, Hui Yao, Tianying Zhang, Yuan Z. Gao, Sijie Zhao, Nan Liu, Weidong Xiao, Yuancai Xiang, Hongming Miao, Liuli Li
| Nature Verified
Abstract Reprogramming of lipid metabolism and cyclic GMP‒AMP synthase (cGAS)-stimulator of interferon genes (STING) signaling is associated with cancer development. However, whether and how fatty acid metabolism regulates the cGAS‒STING pathway in colorectal cancer (CRC) remains to be elucidated. In this study, we found that short-chain acyl-CoA dehydrogenase (ACADS) is aberrantly deficient in CRC cells and is associated with cancer progression in human patients.
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The molecular mechanisms and potential therapeutic implications of the crosstalk between DNA methylation and metabolic reprogramming in thyroid cancer | Cell Death Discovery

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By Tianying Zhang, Hengtong Han, Yating Zhang, Tingting Zhang, Libin Ma, Ze Yang, Yong-xun Zhao
| Nature Verified
Abstract One of the fastest-growing malignant tumors in the world is thyroid cancer (TC), and there are currently no effective treatments for its aggressive subtypes, such as anaplastic carcinoma and radioactive iodine-refractory differentiated thyroid carcinoma. Recent investigations have shown that DNA methylation and metabolic reprogramming are not independent events, but rather create a closely interconnected, mutually reinforcing network of carcinogenic processes.
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