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Trever G. Bivona on Muck Rack

Trever G. Bivona

As seen in: Nature, MDPI, Science Magazine, BioRxiv, Cell Press, Cancer Discovery, Molecular Cancer Research
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Articles

The essential chaperone DNAJC17 activates HSP70 to coordinate RNA splicing and G2-M progression

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By David V Allegakoen, Kristen Kwong, Jacqueline Morales, Trever G. Bivona, Amit J Sabnis, Pushpendra K. Sahu
| Nature Verified
Abstract Molecular chaperones including the heat-shock protein 70-kilodalton (HSP70) family and the J-domain containing protein (JDP) co-chaperones maintain homeostatic balance in eukaryotic cells through regulation of the proteome. The expansive JDP family helps direct specific HSP70 functions, yet loss of single JDP-encoding genes is widely tolerated by mammalian cells, suggesting a high degree of redundancy.
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Temporal photoproximity labeling of ligand-activated EGFR neighborhoods using MultiMap - Nature Chemical Biology

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By Zhi Yuan Lin, Wayne Ngo, Harry X. Wu, Young-wook Jun, Trever G. Bivona, Jennifer Doudna
| Nature Verified
Abstract Photoproximity labeling proteomics (PLP) methods have recently shown that cell surface receptors can form lateral interactome networks. Here, we present a paired set of PLP workflows that dynamically track neighborhood changes for oncogenic epidermal growth factor receptor (EGFR) over time, both outside and inside of cells.
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Acquired resistance in cancer: towards targeted therapeutic strategies - Nature Reviews Cancer

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By Alice Soragni, Erik S. Knudsen, Thomas N. O’Connor, Cristina E. Tognon, Jeffrey W. Tyner, Beatrice Gini, Donghwa Kim, Trever G. Bivona, Xingxing Zang, Agnieszka K. Witkiewicz, David Goodrich, Dadi Jiang, Seth T Gammon, Christopher D. Willey, Paul Boutros, Vlad C. Sandulache, Abdullah Osman, Jeffrey Myers, Kamiya Mehla, Pankaj K. Singh, Keith Chan, Hongbo Gao, Himangi G. Marathe
| Nature Verified
Abstract Development of acquired therapeutic resistance limits the efficacy of cancer treatments and accounts for therapeutic failure in most patients. How resistance arises, varies across cancer types and differs depending on therapeutic modalities is incompletely understood. Novel strategies that address and overcome the various and complex resistance mechanisms necessitate a deep understanding of the underlying dynamics.
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