Xin Hu on Muck Rack

Xin Hu

China
As seen in: Nature, The Oncologist, ACS Publications, Royal Society of Chemistry, Cell Metabolism, Cell Press, Cell Reports Physical Science, The American Journal of Human Genetics, Dove Medical Press, Chemistry World and
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Articles

Complex I protein NDUFB9 is a metabolic vulnerability in triple negative breast cancer brain metastases

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By Mingxi Lin, Zhexu Wen, Cheng Zeng, Yizi Jin, Teng Zhou, Yuxin Yan, Shenglin Huang, Xin Hu, Xiaoxiang Guan, Xichun Hu, Jian Zhang
| Nature Verified
Abstract Triple-negative breast cancer (TNBC) brain metastases (BrMs) remain a therapeutic challenge. We depict the discrepancies between primary tumors and BrMs, and examine patient-matched cerebrospinal fluid and plasma to provide detailed profiles of BrMs’ metabolic microenvironment. High-throughput in vivo loss of function CRISPR screens identify NDUFB9 (NADH: Ubiquinone Oxidoreductase Subunit B9) as a brain-specific metabolic vulnerability.
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Preparation of a pH-Responsive Janus Composite Fibrous Membrane: an Integrated Performance Study on the Visualized Detoxification of a Mustard Gas Simulant and Treatment of Mustard Gas-Induced Skin Injury Click to copy article link Article link copied!

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By Yunfan Cheng, Min Zhang, Xin Hu
| ACS Publications
Abstract Click to copy section linkSection link copied! Mustard gas (HD) causes ulcerative skin wounds; however, owing to its multiple mechanisms of action, no officially approved specific antidote has been developed to date. Moreover, current therapeutic materials for HD-induced skin injuries generally overlook the potential retention of residual HD at the wound site, which may adversely affect the healing process.
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Enhancing platinum-based chemotherapy efficacy and safety through combination therapy-mediated remodeling of autophagic homeostasis in gastric cancer

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By Guangzhao Pan, Qianqian Xu, Kui Zhang, Xin Hu, Chongyang Li, Changhong Li, Haoyan Ji, Xiaosong Hu, Shaomin Shi, Renjian Hu, Chaowei Deng, Erhu Zhao, Jinfei Chen, Hongjuan Cui
| Nature Verified
Abstract Platinum-based drugs exhibit potent anti-tumor efficacy but are limited by low bioavailability, severe toxicity, and resistance. Current therapeutic strategies lack effective solutions due to the unclear molecular mechanisms. Autophagy, with its dual protective/destructive roles, offers potential to enhance platinum-based chemotherapy, yet its clinical translation for optimizing therapeutic outcomes remains underexplored.
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