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Yiling Li

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Articles

Mathematics > Numerical Analysis

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By Yiling Li, Zhiyong Cheng
| arxiv.org
This paper concerns the inverse elastic scattering problem of determining an unknown penetrable obstacle from far-field data. Using the Helmholtz decomposition, the coupled boundary value problem is reformulated as a coupled scalar Helmholtz system. We prove the uniqueness of the associated coupled Helmholtz boundary value problem and of the corresponding boundary integral equation system, which is then discretized by a Nyström method for efficient numerical computation.
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Transcriptional repression by TGIF2 coordinates neurogenic priming and neural stem cell maintenance

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By Pascal J. Schmidt, Neha Chetlangia, Pin-Kuan Chiang, Yiling Li, Anthi Krontira, Franziska Vierl, Maria Richter, Weixu Wang, Juliane Merl-Pham, Fabian J. Theis, Stefanie Hauck, Magdalena Götz
| Science Magazine Verified
Abstract INTRODUCTION RESULTS DISCUSSION MATERIALS AND METHODS Acknowledgments Supplementary Materials REFERENCES Information & Authors Metrics & Citations View Options References Figures Tables Media Share Abstract Stem cells balance self-renewal with differentiation. Priming, i.e., low-level expression of differentiation-related genes, enables stem cells to remain undifferentiated and progeny to fast proceed in up-regulation of differentiation genes.
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Nuclear proteome reveals microtubule-associated protein regulating fate and disease

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By Florencia L. Merino, Lucas Miranda, Aparajita Kumar, Yiling Li, Deepak Kumar Sundaramoorthy, Juliane Merl-Pham, Eva-Maria Schentarra, Clemens Steinek, Veronica Pravatá, Claire M. Kittock, Martina Bürkle, Bob A. Hersbach, Javier Ferri Beneito, Florian Giesert, Ralf Jungmann, Louis-Jan Pilaz, Stefanie Hauck, Silvia Cappello, Magdalena Götz
| Cell Press Verified
Keywords neural stem cells neurogenesis migration cell fate periventricular heterotopia cortical malformations organoids MAP1B moonlighting proteins brain development Introduction Tissue development relies fundamentally on morphogenesis, regulated by the dynamic remodeling of the cytoskeleton as well as transcriptional programs driving cell fate and subtype specification.
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