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Yinan Chen

As seen in: Nature, Wiley Online Library, arXiv, PLOS ONE, MDPI, Taylor & Francis Online, ACS Publications, Science Magazine, Royal Society of Chemistry, BioRxiv and
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Articles

Oriented binding of transcription factors to nucleosomes remodels chromatin at human promoters

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By Tianyi Xiao, Moritz Ochmann, Tushar Warrier, Yepeng Du, Tianyun Hou, Siwei Zou, Yinan Chen, Fangjie Zhu, Dan Zhang, Fatemeh Seyednasrollah, Kimmo Palin, Leena Salmela, Maria Osmala, Sakari Vanharanta, Ekaterina Morgunova, Patrick Cramer, Connor Rogerson, Christian Dienemann, Jussi Taipale, Caillan Crowe- McAuliffe
| Cell Press Verified
ArticleOnline nowOpen access Affiliations & Notes 1Generative Genomics Programme, Wellcome Sanger Institute, Hinxton, United Kingdom of Great Britain and Northern Ireland (the) 2Max-Planck Institute for Multidisciplinary Sciences, Department of Molecular Biology, Göttingen, Germany 3Applied Tumor Genomics Program, Faculty of Medicine, University of Helsinki, Helsinki, Finland 4Haixia Institute of Science and Technology, Fujian Agriculture and Forestry University, Fuzhou, Fujian, China...
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The plural marker tsho in Lhasa Tibetan: Syntax, interpretation, and cross-linguistic analysis

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By Yinan Chen, Yuan Sun, Jie Cheng
| Taylor & Francis Online
ABSTRACT This paper investigates the syntactic distribution and semantic interpretation of the plural marker tsho in Lhasa Tibetan, a topic that has received little attention in the literature. Based on evidence from native-speaker elicitation and previous research, the study argues that tsho should be analyzed as two distinct plural markers, labelled tsho¹ and tsho².
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Single-cell transcriptomics uncovers endothelial progenitor-like remodelling driving human coronary atherosclerosis progression

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By Fang Yao, Fangzhou Li, Shujie Gai, Yinan Chen, Fei Liu, Xingzhong Zhang, Jun Li, Lin Mao, Liliang Guo, Kun Xing, Juan Huang, Shufang Fu, Yang Sun, Jinhua Chen, Heng Zhang, YIHAN CHEN, Bingying Zhou, Li Wang, Zhe Zheng
| Nature Verified
Abstract Coronary atherosclerosis underlies life-threatening conditions such as myocardial infarction and stroke, yet its cellular dynamics remain incompletely understood. Here, through single-cell RNA sequencing of 27,941 cells from 56 human coronary segments, we constructed a disease-stage-resolved cellular atlas, revealing pathological remodelling of endothelial cells (ECs) into a progenitor-like state (EC5SLCO4A1+) with low expression of canonical EC dysfunction signatures.
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