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Ying-Mei Lu

As seen in: Nature, Cell Press
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Articles

Temporal assessment of behavior in Parkinson’s visual hallucinations via a multidimensional analysis strategy

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By Yi-Xuan Zhang, Yun-long Pan, Shu-Yang Chen, Xing-Feng Mao, Li Zhang, Ying-Mei Lu, Feng Han, Quan-Xin Zhang, Chen-Chen Jiang, Jia-Wen Cheng, Shen-Jia Tao, Fukunaga Kohji
| Nature Verified
Abstract Visual hallucination (VH) is a common nonmotor symptom of Parkinson’s disease (PD). However, the lack of reliable animal models and quantitative assessment tools poses significant challenges for mechanism and intervention research in this area. Here, we developed a novel PD-related visual hallucination (PDVH) model by administering benzhexol hydrochloride, which can induce hallucinatory phenotypes in PD model mice.
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Molecular brake on firing pattern transitions in MHb ChAT neurons to mediate nicotine-withdrawal-induced anxiety

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By Zhi-Wei Zheng, Peng-Xiang Min, Yi-Ling Luo, Xing-Feng Mao, Yi-Xuan Zhang, Xiu-Xiu Liu, Ming Lu, Eugene Yujun Xu, Feng Han, Ying-Mei Lu
| Cell Press Verified
Keywords medial habenula cholinergic neurons nicotine withdrawal anxiety burst-firing transition Cav3.1 pumilio 1 Introduction Tobacco addiction remains a global public health crisis. Nicotine, the primary addictive component in tobacco products, induces withdrawal symptoms such as anxiety and irritability upon cessation.
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Endothelial cells-derived SEMA3G suppresses glioblastoma stem cells by inducing c-Myc degradation - Cell Death & Differentiation

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By Yi-Xuan Zhang, Xu Qian, Peng-Xiang Min, Li-Li Feng, Ying-Mei Lu
| Nature Verified
Abstract The poor prognosis of glioblastoma (GBM) patients is attributed mainly to abundant neovascularization and presence of glioblastoma stem cells (GSCs). GSCs are preferentially localized to the perivascular niche to maintain stemness. However, the effect of abnormal communication between endothelial cells (ECs) and GSCs on GBM progression remains unknown. Here, we reveal that ECs-derived SEMA3G, which is aberrantly expressed in GBM patients, impairs GSCs by inducing c-Myc degradation.
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