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Ying Yi

As seen in: Nature, Wiley Online Library, Frontiers, Frontiers in Immunology, MDPI, Taylor & Francis Online, Biomed Central (BMC), BioRxiv, Cell Press, Stem Cell Reports and
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Articles

Glycolytic Reprogramming in Parkinson's Disease: From Bioenergetic Crosstalk to Therapeutic Modulation

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By Ying Yi, Cen Chen, Yu-Zhou Sun
| Wiley Online Library Verified
Conflicts of Interest The authors declare no conflicts of interest. Data Availability Statement Data sharing is not applicable to this article, as no new data were created or analyzed in this study. References 1, , , et al., “Temporal Trends in the Prevalence of Parkinson's Disease From 1980 to 2023: A Systematic Review and Meta-Analysis,” Lancet Healthy Longevity 5, no. 7 (2024): e464–e479, https://doi.org/10.1016/s2666-7568(24)00094-1.
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Regulation of Extracellular HMGB-1 Alarmin Levels by CIGB-300 Anticancer Peptide In Vitro e In Vivo

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By Ying Yi, Daylen Aguilar-Noriega, Jamilet Miranda
| Preprints.org
You are already at the latest version HMGB-1 is an alarmin representative of DAMP playing a central role in the immuno-genic cell death (ICD), a necessary condition in the dialogue stablished between dying tumor cells and immune system during some anticancer therapies. Therefore, early screening for ICD inducers represents a major priority in drug development today.
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NAT10 as a central node in cancer biology: integrating epitranscriptomic regulation, metabolic reprogramming, and immune modulation

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By Wentao Bo, Ying Yi, Biao Zhao, Hang Dong
| Frontiers in Immunology
Abstract N-acetyltransferase 10 (NAT10), the sole known mRNA N4-acetylcytidine (ac4C) writer, has emerged as a central regulator of cancer adaptation. Beyond its canonical role in RNA acetylation, NAT10 integrates environmental stress signals—including hypoxia, metabolic imbalance, and inflammatory cues—with epitranscriptomic remodeling to sustain malignant progression.
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