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Yingying Pu

As seen in: Nature, PLOS, Cell Press
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Articles

Hijacking of host PCNA by circovirus replication-associated protein to recruit POLD1 drives viral DNA replication and is inhibited by R428

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By Lei Zhu, Guan Zhang, Yingying Pu, PLOS Pathogens
| PLOS
Loading metrics Open Access Peer-reviewed Research Article Citation: Zhu L, Zhang G, Pu Y, Wang W, Zhang Z, Kang J, et al. (2026) Hijacking of host PCNA by circovirus replication-associated protein to recruit POLD1 drives viral DNA replication and is inhibited by R428. PLoS Pathog 22(9): e1014575. https://doi.org/10.1371/journal.ppat.1014575 Editor: Jeremy P.
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SNRPD2–CPSF7–UBE2K axis drives ovarian cancer progression via alternative splicing–polyadenylation crosstalk

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By Yingwei Li, Zhongshao Chen, Qianqian Gao, Yuehan Gao, Yingying Pu, Ning Yang, Yuchao Diao
| Nature Verified
Abstract Aberrant alternative polyadenylation (APA) and alternative splicing (AS) contribute to numerous diseases, including cancer; however, their coordinated roles in ovarian cancer remain poorly understood. Here, we investigated CPSF7, an APA factor markedly upregulated in ovarian cancer and associated with poor prognosis.
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Spliceosomal component SNRPE drives cell proliferation by regulating CTP synthase 1 mRNA splicing in ovarian cancer

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By Yingying Pu, Zhongshao Chen, Qianqian Gao, Yanling Liu, Ning Yang, Yingwei Li, Beihua Kong
| Nature Verified
Abstract Sustained proliferation is a hallmark of tumor cells. Cancer-associated alternative splicing (AS) events can provide proliferative advantages in tumors, suggesting that identifying aberrant RNA splicing events linked to proliferation in ovarian cancer (OC) may reveal novel therapeutic targets. In this study, we found that small nuclear ribonucleoprotein polypeptide E (SNRPE) was overexpressed in OC, particularly in the proliferative subtype, and indicated worse clinical prognosis.
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