Yingying Zeng
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As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.Articles
Learning from Finland: NGO Perspectives on Empowering Immigrants with Financial Capability
Abstract This perspective paper draws on field notes and practitioner voices to explore how NGO professionals in Finland understand and support financial capability among immigrant populations. Financial capability—encompassing knowledge and access to financial tools—is essential for immigrant integration and stability.
Disease-associated microglia adopt stage-specific phenotypes that regulate T cell fate and immunity in glioma
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Keywords microglia disease-associated microglia DAMs macrophage glioma myeloid cells microglia-T cell interaction MHC class II PD-L1 Get full text access Log in, subscribe or purchase for full access. References 1. Louis, D.N. ∙ Perry, A. ∙ Wesseling, P. ... The 2021 WHO Classification of Tumors of the Central Nervous System: a summary Neuro. Oncol. 2021; 23:1231-1251 2. Zhang, J. ∙ Mu, R. ∙ Liu, F. Advances in brain tumor therapy: from molecular diagnostics to novel treatments Sci. China Life Sci.
By Jiawen Qian, Chen Wang, Yi Zhang, Yingying Zeng, Shuangyan Zhang, Fei Gao, Yiwen Zhu, Peiliang Cai, Zhongyuan Zhang, Yanan Hu, Xiaotong Yu, Benoît Kloeckner, Marco Moreira, Yuedi Wang, Bo Wang, Chujun Zhao, Dan Zhang, Xi Chen, Shang Liu, Ronghua Liu, Feifei Luo, Liang Wu, Claudia Pasqualini, Yihua Chen, Xinyan Li, Mingfang Lu, Ziyi Li, Wei Guo, Svetoslav Chakarov, Zhaoyuan Liu, Bing Su, Rui Zhao, Yiwei Chu, Florent Ginhoux, Congwen Li, Auriane Coutansais, Huangmijia Zhuang
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Cell Press
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G3BP2 confers venetoclax resistance by stabilizing ELF1-mediated MCL1 transcription in acute myeloid leukemia
Abstract Venetoclax is a promising treatment for acute myeloid leukemia (AML) patients, but resistance occurs following the upregulation of MCL1. The RNA-binding protein G3BP2 is upregulated in various solid tumors, and targeting G3BP2 has been shown to restore chemosensitivity. However, the role of G3BP2 in AML development and venetoclax resistance remains unknown. Herein, an integrated analysis revealed that G3BP2 confers venetoclax resistance and correlates with poor outcomes in AML.
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