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Yunkyoung Lee

As seen in: Nature, MDPI, The Journal of Biological Chemistry
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Articles

Hepatocyte TonEBP promotes metabolic stress-induced hepatic fibroinflammation involving transcriptional activation of ELR⁺ CXC chemokines - Cell Death Discovery

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By Jun Lee, Eun Jin Yoo, Yeseul Jeong, Gee Euhn Choi, Youngheun Jee, MinHyeok Kang, Jiwon Yang, Sung-Pyo Hur, Jong-Eun Park, Yunkyoung Lee, Hye-Kyung Park, Ji-Hyun Yun, Whaseon Lee-Kwon, Hyug Moo Kwon, Soo Youn Choi, Hana Song, Seung Mi Ko, Mi-Kyoung Jang
| Nature Verified
Abstract Metabolic dysfunction-associated steatohepatitis (MASH), a progressive stage of metabolic dysfunction-associated steatotic liver disease (MASLD), is characterized by liver inflammation, fibrosis, and hepatocyte injury. Despite its clinical relevance, the molecular mechanisms linking metabolic stress to hepatic fibroinflammation remain poorly understood.
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Integrating microbiome and metabolome analyses to unravel the role of inulin in enhancing the meat quality and bone health of ducks - Scientific Reports

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By Yunkyoung Lee
| Nature Verified
Abstract Inulin, a natural dietary fiber, has various health-promoting bioactivities. However, the impacts of dietary inulin on duck productivity and overall health remain unclear. This study investigated the effects of inulin supplementation on systemic health and its underlying mechanisms by integrating the microbiota and metabolome analyses.
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Lipopolysaccharide (LPS)-stimulated iNOS Induction Is Increased by Glucosamine under Normal Glucose Conditions but Is Inhibited by Glucosamine under High Glucose Conditions in Macrophage Cells

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By Ji-Sun Hwang, Mi-Youn Kwon, Kyung-Hong Kim, Yunkyoung Lee
| The Journal of Biological Chemistry Verified
We investigated the regulatory effect of glucosamine (GlcN) for the production of nitric oxide (NO) and expression of inducible NO synthase (iNOS) under various glucose conditions in macrophage cells. At normal glucose concentrations, GlcN dose dependently increased LPS-stimulated production of NO/iNOS. However, GlcN suppressed NO/iNOS production under high glucose culture conditions. Moreover, GlcN suppressed LPS-induced up-regulation of COX-2, IL-6, and TNF-?
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