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Zuberwasim Sayyad on Muck Rack

Zuberwasim Sayyad

As seen in: Nature, MDPI, BioRxiv, Cell Press
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Articles

Virus-induced deconjugation of ISG15 dysregulates innate immunity and cellular metabolism

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By Junji Zhu, GuanQun Liu, Jielin Xu, Zuberwasim Sayyad, Kun Li, Surajit Chakraborty, Christopher Goins, Huaxu Yu, Yadi Zhou, Shanti Ross, Evangeline White, Jun Zhao, Oliver Fiehn, Shaun Stauffer, Feixiong Cheng, Michaela U. Gack
| Cell Press Verified
ArticleOnline nowOpen access Affiliations & Notes 1Florida Research and Innovation Center, Cleveland Clinic, Port Saint Lucie, FL, 34987, United States of America 2Department of Genomic Sciences and Systems Biology, Cleveland Clinic Research, Cleveland Clinic, Cleveland, OH, 44195, United States of America 3Cleveland Clinic Genome Center, Cleveland Clinic Research, Cleveland Clinic, Cleveland, OH, 44195, United States of America 4Center for Therapeutics Discovery, Cleveland Clinic Research,...
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Publisher Correction: TRIM23 mediates cGAS-induced autophagy in anti-HSV defense - Nature Communications

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By Zuberwasim Sayyad, Junji Zhu, Jiro Kato, Shen-Ying Zhang
| Nature Verified
Correction to: Nature Communications https://doi.org/10.1038/s41467-025-59338-5, published online 13 May 2025 In this article the wrong figure appeared as Fig. 2.; the figure should have appeared as shown below. The original article has been corrected.
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TRIM23 mediates cGAS-induced autophagy in anti-HSV defense - Nature Communications

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By Zuberwasim Sayyad, Junji Zhu, Jiro Kato, Shen-Ying Zhang
| Nature Verified
Abstract The cGAS-STING pathway, well-known to elicit interferon (IFN) responses, is also a key inducer of autophagy upon virus infection or other stimuli. Whereas the mediators for cGAS-induced IFN responses are well characterized, much less is known about how cGAS elicits autophagy. Here, we report that TRIM23, a unique TRIM protein harboring both ubiquitin E3 ligase and GTPase activity, is crucial for cGAS-STING-dependent antiviral autophagy.
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