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Shen-Ying Zhang

As seen in: Nature, Science Magazine, Cell Press
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Articles

Publisher Correction: TRIM23 mediates cGAS-induced autophagy in anti-HSV defense - Nature Communications

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By Zuberwasim Sayyad, Junji Zhu, Jiro Kato, Shen-Ying Zhang
| Nature Verified
Correction to: Nature Communications https://doi.org/10.1038/s41467-025-59338-5, published online 13 May 2025 In this article the wrong figure appeared as Fig. 2.; the figure should have appeared as shown below. The original article has been corrected.
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TRIM23 mediates cGAS-induced autophagy in anti-HSV defense - Nature Communications

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By Zuberwasim Sayyad, Junji Zhu, Jiro Kato, Shen-Ying Zhang
| Nature Verified
Abstract The cGAS-STING pathway, well-known to elicit interferon (IFN) responses, is also a key inducer of autophagy upon virus infection or other stimuli. Whereas the mediators for cGAS-induced IFN responses are well characterized, much less is known about how cGAS elicits autophagy. Here, we report that TRIM23, a unique TRIM protein harboring both ubiquitin E3 ligase and GTPase activity, is crucial for cGAS-STING-dependent antiviral autophagy.
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Genetic defects of brain immunity in childhood herpes simplex encephalitis - Nature

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By Shen-Ying Zhang, Jean-Laurent Casanova Verified
| Nature Verified
Abstract Herpes simplex virus 1 (HSV-1) encephalitis (HSE) is the most common sporadic viral encephalitis in humans. It is life-threatening and has a first peak of incidence in childhood, during primary infection. Children with HSE are not particularly prone to other infections, including HSV-1 infections of tissues other than the brain.
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