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Aatmika Barve

As seen in: BioRxiv, Cell Reports Medicine
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Articles

Plasma growth-associated protein 43 correlates with synaptic loss in Alzheimer’s disease

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By Guoyu Lan, Binyin Li, Mengjie Wang, Aatmika Barve, Morvane Colin, Luc Buée, Laihong Zhang, Mingxing Jiang, Jie Yang, Anqi Li, Zhengbo He, Xin Zhou, Yalin Zhu, Yue Cai, Pan Sun, Lin Liu, Linting Chen, Lili Fang, Yiying Wang, Mingxu Li, Xuhui Chen, Dai Shi, Chenghui Ye, Xiang Fan, Qingyong Wang, Liemin Zhou, Zhen Liu, Ying Han, Lu Wang, Guanxun Cheng, Yihui Guan, Ruiqing Ni, Kevin Richetin, Fang Xie, Tengfei Guo, Jieyin Li
| Cell Reports Medicine Verified
Keywords synaptic loss blood biomarkers GAP-43 extracellular vesicles neuropathology Alzheimer’s disease Introduction Dementia is the leading cause of disability and death among older adults and currently affects over 55 million people worldwide.1 Approximately 60%–70% of dementia cases are attributed to Alzheimer’s disease (AD), marked by the deposition of extracellular amyloid-β (Aβ) plaques and intraneuronal tau tangles composed of phosphorylated tau (p-tau), and then dementia with Lewy...
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Reduced levels of synaptic vesicle protein 2A in the extracellular vesicles and brain of Alzheimer′s disease- associations with Aβ, tau and synaptophysin

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By Aatmika Barve, Valentin Zufferey, Jeanne Espourteille, Jana Nussbaumer
| BioRxiv
Thank you for your interest in spreading the word about bioRxiv. NOTE: Your email address is requested solely to identify you as the sender of this article. Your Email * Your Name * Send To * Enter multiple addresses on separate lines or separate them with commas. Message Subject (Your Name) has forwarded a page to you from bioRxiv Message Body (Your Name) thought you would like to see this page from the bioRxiv website.
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Astrocyte and mitochondrial footprints in brain-derived extracellular vesicles predict tau pathology

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By Jeanne Espourteille, Aatmika Barve, Valentin Zufferey, Elodie Leroux
| BioRxiv
Abstract Tauopathies are neurodegenerative disorders characterized by abnormal tau aggregation, with primary 3R (e.g., Picks disease, PiD) and 4R (e.g., progressive supranuclear palsy, PSP) variants posing a significant diagnostic challenge. Here, we examined brain-derived extracellular vesicles (BD-EVs) isolated from the prefrontal cortex of PiD (3R), PSP (4R), and non-demented controls (CTRL) to determine if these vesicles reflect disease-specific proteomic signatures.
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