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ArticleVolume 85, Issue 24p4492-4509.e11Open access 1University of Toulouse UT, CNRS, CBI, MCD, Toulouse, France 2University of Toulouse UT, CNRS, CBI, BigA, NGS Data Analysis Facility, Toulouse, France 3University of Toulouse UT, CNRS, CBI, LITC Core Facility, Toulouse, France 4University of Toulouse UT, CNRS, Institut de Pharmacologie et de Biologie Structurale (IPBS), Toulouse, France 5Equipe Labellisée Ligue Contre le Cancer 2018, Toulouse, France 6Institut National de la Santé et de la...
Abstract RNA:DNA hybrids accumulate at DNA double-strand breaks (DSBs) and were shown to regulate homologous recombination repair. The mechanism responsible for the formation of these non-canonical RNA:DNA structures remains unclear although they were proposed to arise consequently to RNA polymerase II or III loading followed by DSB-induced de novo transcription at the break site.
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