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Barbara Martinez-Pastor

As seen in: Nature, PLOS ONE, Cell Reports
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Articles

Publisher Correction: Polyamine-dependent metabolic shielding regulates alternative splicing - Nature

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By Amaia Zabala-Letona, Maria Ponce-Rodriguez, Saioa Garcia-Longarte, Isabel Mendizabal, Malgorzata Ewa Rogalska, Joycelyn Tan, Pilar Ximenez-Embun, Maciej Zakrzewski, Ianire Astobiza, Sarah Cherkaoui, Maria Sendino, Amaia Ercilla, Laura Bozal-Basterra, Encarnación Pérez-Andrés, Juan A. Ferrer-Bonsoms, Maciej Cieśla, Joan Seoane, Rosa Barrio, James Sutherland Verified, Ana M. Aransay, Juan Manuel Falcon-Pérez, Barbara Martinez-Pastor, Francisco Blanco, Michael D. Hogarty, Raphael J. Morscher, Remigiusz A. Serwa, Jesús Jiménez-Barbero, Gonzalo Jiménez-Osés, Alejo Efeyan, Lydia W. S. Finley, Javier Muñoz, Juan Valcárcel
| Nature Verified
Correction to: Nature https://doi.org/10.1038/s41586-025-09965-1 Published online 14 January 2026 In the version of the article initially published, in Fig. 1g, the three right-hand labels reading “24 h” should have read “SAT1”, “SMARCA1” and “ACTB”. This correction has been made to the HTML and PDF versions of the article, as seen in Fig. 1, below. Fig. 1: Original and corrected Fig.
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Polyamine-dependent metabolic shielding regulates alternative splicing - Nature

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By Amaia Zabala-Letona, Saioa Garcia-Longarte, Isabel Mendizabal, Malgorzata Ewa Rogalska, Joycelyn Tan, Pilar Ximenez-Embun, Maciej Zakrzewski, Ianire Astobiza, Sarah Cherkaoui, Maria Sendino, Amaia Ercilla, Laura Bozal-Basterra, Juan A. Ferrer-Bonsoms, Joan Seoane, Rosa Barrio, James Sutherland Verified, Ana M. Aransay, Juan Manuel Falcon-Pérez, Barbara Martinez-Pastor, Francisco Blanco, Michael D. Hogarty, Raphael J. Morscher, Remigiusz A. Serwa, Jesús Jiménez-Barbero, Gonzalo Jiménez-Osés, Alejo Efeyan, Lydia W. S. Finley, Javier Muñoz, Juan Valcárcel, Maria Ponce-Rodriguez, Encarnación Pérez-Andrés, Maciej Cieśla
| Nature Verified
Abstract Metabolites are central to cellular homeostasis. Although much emphasis has been placed on their relevance to meet energetic and biosynthetic demands, metabolic intermediates also function as signalling molecules. Here we show that polyamines, small polycations that are critical to cellular homeostasis1,2,3, regulate the process of alternative pre-mRNA splicing.
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ZNF280A links DNA double-strand break repair to human 22q11.2 distal deletion syndrome - Nature Cell Biology

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By Thomas Clarke, Ilaria Ceppi, Giorgia G. Silveira, Ruben Boon, Barbara Martinez-Pastor, Tiziano Bernasocchi, Lee Zou, Donna McDonald-McGinn, Petr Cejka, Zeeba Kamaliyan, Blaine T. Crowley
| Nature Verified
Abstract DNA double-strand breaks (DSB) are among the most deleterious forms of DNA damage and, if unresolved, result in DNA mutations and chromosomal aberrations that can cause disease, including cancer. Repair of DSBs by homologous recombination requires extensive nucleolytic digestion of DNA ends in a process known as DNA-end resection.
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