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Jooman Park

As seen in: Nature, Wiley Online Library, Science Magazine, BioRxiv, Cell Press
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Articles

ERα activates NAMPT/IL-33 signaling to enhance beige thermogenesis and metabolic fitness

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By Janni Yuval, Xintao Li, Juha Aalto, Ruoci Hu, Jooman Park, Yanyu Qian, Shaolei Xiong, Aditya Chhikara, Zuoxiao Shi, Lifeng Liu, Yanhui Li, Zhenyuan Song, Abeer M. Mahmoud, Jiwang Chen, Joseph A. Baur, Yanlin He, Brian T. Layden, Zhenqi Zhou, Pingwen Xu, Ahmad Hamza Elsabbagh, Huailing Fan
| Science Magazine Verified
Abstract Beige adipocytes are inducible thermogenic fat cells that emerge within white adipose tissue (WAT) in response to thermogenic stimuli and confer metabolic benefits. However, obesity impairs the generation of beige adipocytes, and the underlying mechanisms remain poorly understood. Here, we show that obesity leads to a loss of adipose progenitor cells (APCs) in WAT, accompanied by reduced estrogen (E2) levels and nicotinamide phosphoribosyltransferase (NAMPT) expression.
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Targeting Adipose Tissue Function Protects Against Heart Failure with Preserved Ejection Fraction

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By Jordan Jousma, Zhenbo Han, Jooman Park, Gege Yan
| Wiley Online Library Verified
1 Introduction Heart failure (HF) remains a leading cause of death and disability worldwide.[1] In recent years, the prevalence of heart failure with preserved ejection fraction (HFpEF) has rapidly increased and now comprises approximately half of all HF cases.
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Skeletal muscle endothelial dysfunction through the activin A–PGC1α axis drives progression of cancer cachexia - Nature Cancer

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By Young-Mee Kim, Mark Sanborn, Shaluah Vijeth, Priyanka Gajwani, Dahee Jung, Georgina E Mancinelli, Peter T Toth, Evan Phillips, Paul J. Grippo, Ameen A. Salahudeen, Jooman Park, Su Yeon, Yuwei Jiang, Steve Lee
| Nature Verified
Abstract Cachexia is the wasting of skeletal muscle in cancer and is a major complication that impacts a person’s quality of life. We hypothesized that cachexia is mediated by dysfunction of the vascular system, which is essential for maintaining perfusion and tempering inappropriate immune responses.
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