Mark Sanborn on Muck Rack

Mark Sanborn

Denver
As seen in: LinkedIn, Medium, Nature, Entrepreneur, Fast Company, Cell Press, Nigerian Tribune, Life Science Leader, Farm Equipment, Gulfshore Business and
Inspirational leadership keynote speaker and best selling author. Learn how to lead the future workforce now (new research based presentation).
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Articles

Ionic regulation of cancer cell stiffness and metastatic colonization via the MRTFA-KCNMB1 axis

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By Alexa M Gajda, Mohamed Haloul, Khushi Patel, Raymundo Rodríguez-López, Katie Beverley, Mark Sanborn, Kihak Lee, Stephanie M Wilk, Beata M. Wolska, Faruk Hossen, James Lee, Irena Levitan, Jalees Rehman, Ekrem Emrah Er, Keyvan Mollaeian, Caitlyn C. Castillo, Eron N. Mendenhall
| Cell Press
Keywords MRTFA SRF KCNMA1 KCNMB1 cancer metastasis cell stiffness ion channels mechanobiology mechanosurveillance Introduction Cellular stiffness is a dynamically regulated property that allows cells to deform with external force and respond to biochemical and mechanochemical stimuli in the microenvironment.1 Maintenance and regulation of cellular stiffness and mechanoadaptation play key roles in various physiological and pathological states.
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Human hippocampal neurogenesis in adulthood, ageing and Alzheimer’s disease

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By Ahmed Disouky, Mark Sanborn, K. R. Sabitha, Mostafa Mostafa, Ivan Ayala, David Bennett, Yisha Lu, Yi Zhou, C. Dirk Keene, Sandra Weintraub, Tamar Gefen, M.-Marsel Mesulam, Changiz Geula, Mark Maienschein-Cline, Jalees Rehman, Orly Lazarov
| Nature
Abstract The existence of human hippocampal neurogenesis has long been disputed1,2,3,4,5,6,7,8,9,10,11,12 and its relevance in cognition remains unknown. Recent studies have established the presence of proliferating progenitors and immature neurons and a reduction in the latter in Alzheimer’s disease (AD)11,13. However, their origin and the molecular networks that regulate neurogenesis and function are poorly understood.
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Skeletal muscle endothelial dysfunction through the activin A–PGC1α axis drives progression of cancer cachexia - Nature Cancer

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By Young-Mee Kim, Mark Sanborn, Shaluah Vijeth, Priyanka Gajwani, Dahee Jung, Georgina E Mancinelli, Peter T Toth, Evan Phillips, Paul J. Grippo, Ameen A. Salahudeen, Jooman Park, Su Yeon, Yuwei Jiang, Steve Lee
| Nature
Abstract Cachexia is the wasting of skeletal muscle in cancer and is a major complication that impacts a person’s quality of life. We hypothesized that cachexia is mediated by dysfunction of the vascular system, which is essential for maintaining perfusion and tempering inappropriate immune responses.
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