Skip To Main Content
K. W. Annie Bang on Muck Rack

K. W. Annie Bang

As seen in: Nature, Diabetes Magazine
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with K. W. Annie

Contact K. W. Annie, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

Aneuploidy selects for the acquisition of driver genes in breast cancer

|
By Khalid N Al-Zahrani, Jocelyn Nurtanto, Andreea Obersterescu, Katie Teng, Christopher Lowden, Lauren Caldwell, Miguel A. Pérez-Castro, Ricky Tsai, Alexander T Bahcheli, Shifei Wu, Masahiro Narimatsu, Iosifina Fotiadou, Kin Chuah Chan, Linkang Zhang, K. W. Annie Bang, Michael Parsons, Larissa Mourao, Palavalasa Sravya, Li Zhang, Peter Sajjakulnukit, Costas A. Lyssiotis, Alexander D. Borowsky, Colinda L. G. J. Scheele, Daniel Wahl, Hartland W. Jackson, Katherine Stewart, Elaine Fuchs, Sean Egan, Miguel Angel Pujana, Jüri Reimand, Jeffrey L. Wrana, Daniel Schramek, Ellen R. Langille, Julien Dessapt, Cynthia H. Chiu, Geraldine Mbamalu, Adele G. Lopes, E. Idil Temel, Liddy McCulla
| Nature Verified
Abstract Chromosome instability is highly prevalent in cancer and drives large-scale chromosomal imbalances, known as aneuploidies1,2,3,4. How aneuploidy contributes to tumorigenesis remains difficult to study due to the vast numbers of genes affected. Here we established a CRISPR knockout- and activation-linked assay (CRISPR-KOALA), enabling high-throughput bidirectional genetic screens in immunocompetent mouse models of cancer.
Open in Who Shared Issue with article?
|
By Erin E. Mulvihill, Elodie M. Varin, Jonathan E. Campbell, John R. Ussher, Jennifer Ayala, K. W. Annie Bang, Daniel J. Drucker, Bojana Gladanac, Laurie L. Baggio, Bernardo Yusta, Melissa A. Burmeister, Dianne Matthews, Julio E. Ayala
| secure.jbs.elsevierhealth.com
Open in Who Shared Issue with article?

Inhibition of Dipeptidyl Peptidase-4 Impairs Ventricular Function and Promotes Cardiac Fibrosis in High Fat-Fed Diabetic Mice

|
By Erin E. Mulvihill, Elodie M. Varin, John R. Ussher, Jonathan E. Campbell, K. W. Annie Bang
| Diabetes Magazine Verified
Dipeptidyl peptidase-4 (DPP4) inhibitors used for the treatment of type 2 diabetes are cardioprotective in preclinical studies; however, some cardiovascular outcome studies revealed increased hospitalization rates for heart failure (HF) among a subset of DPP4 inhibitor–treated subjects with diabetes.
Open in Who Shared Issue with article?
See all 3 articles