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Kaitlin M. Budd

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Articles

Dominant clones leverage developmental epigenomic states to drive ependymoma

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By Alisha Kardian, Hua Sun, Siri Ippagunta, Nicholas Laboe, Srinidhi Varadharajan, Kwanha Yu, Hsiao-Chi Chen, Tuyu Zheng, Jon P. Connelly, Yong-Dong Wang, Jiangshan Zhan, Hengxi Liu, Kimberley Lowe, Taylor Bugbee, Rakesh Pathak, Amanda Bland, Sanya Mehta, Amir Arabzade, Blake Holcomb, Tristen Wright, David S. Gee, Yi Xiao, Samuel K. McBrayer, Thomas E. Merchant, Jun Qi, Adam D. Durbin Verified, Lindsay A. Schwarz, Li Wang, Andrew M Donson, Nicholas Foreman, Sameer Agnihotri, Alfonso Lavado, Suzanne Baker, David Ellison, Hyun Lee, Shondra M. Pruett-Miller, Kelsey C. Bertrand, Benjamin Deneen, Stephen Mack, Erik Emanus, Riley M. Deneen, Sophie Cochiolo, Kaitlin M. Budd, Gabriele Kembuan, Emma Caesar, Amelia Hancock, Joel Murdoch
| Nature Verified
Abstract ZFTA–RELA is the most recurrent genetic alteration seen in paediatric supratentorial ependymoma (EPN) and is sufficient to initiate tumours in mice1. Despite its oncogenic potential, ZFTA–RELA (ZR) is observed nearly exclusively in childhood EPN, with tumours located distinctly in the supratentorial brain of the central nervous system1.
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