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Lianying Jiao on Muck Rack

Lianying Jiao

As seen in: Nature, PNAS, Science Magazine, Cell Press
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Articles

Pregnancy promotes tumor metastasis through adenosine-induced immunosuppressive neutrophils in pre-metastatic microenvironment

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By CHEN CHEN, Jiahong Shi, Yutiantian Lei, Qian Wang, Peihai Cao, Dairu Hou, Mengdi Huang, Ruiying Wang, Qiuyu Mu, Tingting Lv, Rui Jia, Mingjun Si, Dingge Jiang, Zhixuan Cai, Yakun Huang, Jiahao Sun, Lianying Jiao, Zhengtao Xiao, Yuhan Li, Binxian Wang, Shan Huang, Wenqian Du, Lu Bai, Jinlu Ma, Suxia Han, Baojun Zhang, Chen Lin Verified, Hui Guo, Xiangjiao Meng, Yuzhu Hou
| Nature Verified
Abstract Tumor metastasis is the primary cause of cancer treatment failure and mortality. Pregnant patients with cancer sometimes experience a poor prognosis and accelerated disease progression, yet the underlying mechanisms remain poorly understood. Here we show that pregnancy enhances tumor metastasis in female mice by elevating adenosine levels, which drives pre-metastatic niche remodeling.
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TUFT1 stabilizes TGF-β receptor II protein and facilitates activation of hepatic stellate cells into metastasis-promoting myofibroblasts - Cell Death & Differentiation

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By YUE LI, Yu Shi, Yaxin Fu, Lianying Jiao, Hanqi Li, Lu Shao, Xuelian Xiao, Ningling Kang, Liankang Sun
| Nature Verified
Abstract Cancer-associated fibroblasts (CAFs) transdifferentiated from hepatic stellate cells (HSCs) are a critical determinant of liver metastasis of colorectal cancer (CRC). However, the mechanisms behind transforming growth factor β (TGF-β)-stimulated activation of HSCs into CAFs remain poorly understood. Immunoprecipitation coupled with mass spectrometry identified tuftelin 1 (TUFT1) as a novel TGF-β receptor II (TβRII) binding protein in primary human HSCs and immortalized LX2 cells.
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Calcium-sensitive synaptotagmin 11-lipid interaction modulates exo-endocytosis - Nature Communications

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By Xuanang Wu, Cong Ma, Ying Lai, Lianying Jiao, Changhe Wang
| Nature Verified
Abstract Synaptotagmins (Syts) are the primary Ca2+-sensors for synaptic vesicle exocytosis, while most mammalian Syts are non-Ca2+-affinitive and play critical roles in neurotransmission and synaptic plasticity with unclear mechanisms. Here, we show that high-alkaline non-Ca2+-binding Syt11 exhibits higher affinity for acidic phospholipids and Ca2+-inhibited liposome-binding, thereby competing with the Ca2+-binding Syt1.
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