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Lisa Dyer

As seen in: U.S. Department of State, Court Watch with Alex Swoyer, Cell Press, The American Journal of Human Genetics, SpaceNews, Breaking Defense, San Antonio Express-News, The Salem News (Salem, MA), Data Center Dynamics, Frederick News Post and

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Articles

De novo variants in LDB1 are linked to distinct neurodevelopmental phenotypes determined by variant location and differing pathomechanisms

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By Mireia Coll-Tané, Theresa Brunet, Benjamin Cogné, Mathilde Nizon, Francesco Nicita, Lorena Travaglini, Antonio Novelli, Amanda Peterson, Jillian G. Buchan, Kolja Meier, Jutta Gärtner, Susann Diegmann, Véronique Pingault, Tania Attié-Bitach, Thomas Courtin, Michael Schneider, Lauren O’Grady, Katharina Steindl, Sarju G. Mehta, Christel Depienne, Boris Keren, Solveig Heide, Shane McKee, Franco Laccone, Lisa Dyer, Wendy Jones, Divya Vats, Christiane Zweier, Heinrich Sticht, Anne Gregor, Rebecca Fluri, Solene Conrad, Margie Glissmeyer, Dan Serber, Inderneel Sahai, Catherine Melver, Connie Motter, Zoey Trueblood Wilson, Kristina Huß
| The American Journal of Human Genetics Verified
Keywords neurodevelopmental disorder Drosophila melanogaster rare disease disease gene discovery Introduction LIM domain binding gene 1 (LDB1; also known as NLI and CLIM2) (MIM: 603451) encodes a ubiquitously expressed co-regulator of transcription.1 Studies in mice have shown its importance in neurogenesis, including development of the fore-,1 mid- and hindbrain.2 Additionally, it plays an essential role in the differentiation of corticospinal motoneurons and interneurons,3 axon guidance,4...
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De novo variants in KDM2A cause a syndromic neurodevelopmental disorder

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By Eric Anderson, Stephan Holger Drukewitz, Sukhleen Kour, Deepa Rajan, Karen Stals, Deirdre Donnelly, Tiong Y. Tan, Zornitza Stark, Pia Zacher, Nicolas Chatron, Séverine Drunat, Yoann Vial, Xenia Latypova, Jonathan Levy, Alain Verloes, Jennefer N. Carter, Devon E. Bonner, Suma P. Shankar, Jonathan Bernstein, Julie Cohen, Anne M. Comi, Deanna Alexis Carere, Lisa Dyer, Sureni V. Mullegama, Pedro A. Sanchez-Lara, Katheryn Grand, Hyung-Goo Kim, Afif Ben-Mahmoud, Sidney M. Gospe, Gary Bellus, Renske Oegema, Anita Rauch, Ivan Ivanovski, Frédéric Tran Mau-Them, Aurore Garde, Rachel A. Rabin, John Pappas, Annette E. Bley, Timo Wagner, Eva L. Decker, Carsten Bergmann Verified, Henri Margot, Johannes R. Lemke, Rami Abou Jamra, Julia Hentschel, Heather C Mefford, Amit Singh, Udai Bhan Pandey, Konrad Platzer, Anuradha V. Chimata, Senta Schönnagel, John F. Mantovani, Pauline Monin, Rebecca S. Belles, Klaske D. Lichtenbelt, Janna Bredow, Louis Domenach
| The American Journal of Human Genetics Verified
Keywords KDM2A neurodevelopmental disorder epigenetic machinery Introduction The epigenetic machinery encompasses proteins that function as writers, erasers, readers, and remodelers of epigenetic marks on DNA and histones. The eraser KDM2A removes mono- and di-methylation from histone 3 at lysine 36 (H3K36).
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Lisa Dyer: Listen to Whispers, Not Shouts

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By Lisa Dyer
| Broadband Breakfast Verified
A wise person once said, “Listen to whispers, not shouts.” The March 2025 Federal Communications Commission Notice of Inquiry on Positioning, Navigation and Timing (PNT) prompted numerous shouts among stakeholders, with a time-worn theme emerging: jamming and spoofing vulnerabilities of the Global Positioning System (GPS) require a wholesale change to our PNT strategy. The loudest proponents were companies with something to sell to support this dramatic shift.
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