Pierre Bertrand on Muck Rack

Pierre Bertrand

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Barcelona
As seen in: The Wall Street Journal, MSN, Nature, Yahoo News, Canadian Broadcasting Corporation (CBC), The London Standard, The Wall Street Journal (China), Yahoo Finance, Euronews, Libération, MarketWatch and
Reporter, Dow Jones Newswires This is my actual account. Messages from an "H @PierreBtrand" aren't from me. It has been repoted.
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Articles

Hypusination of the translation factor eIF5A regulates mitochondrial tRNA processing to promote prostate cancer aggressiveness

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By Marc Pujalte-Martin, Anne Vincent, Loïc Trapani, Paraskevi Kousteridou, Amine Belaid, Emilie Thomas, Pierre Bertrand , Heather Carr, Frédéric Larbret, Emilie Baudelet, Stéphane Audebert, Didier F. Pisani, Luc Camoin, Mathieu Carlier, Matthieu Durand, Damien Ambrosetti, Yu Chen, Jean-Jacques Diaz, Arnaud Jacquel, Issam Ben-Sahra, Frederic Bost, Michel Kahi, Ludovic Batistic, Victor Tiroille, Joseph Murdaca, Oumayma Benaceur, Marie Irondelle, Christelle Boscagli, Silvère Baron, Nathalie M. Mazure, Pascal Peraldi
| Nature
Abstract Protein synthesis plays a central role in cancer development and progression. eukaryotic initiation factor 5 A (eIF5A), a translation factor activated by hypusination, is implicated in tumorigenesis, however, its mode of action is still unclear. We find that hypusinated eIF5A (eIF5Ahyp) promotes metastasis and tumor growth in prostate cancer (PCa) by supporting mitochondrial metabolism and translation.
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Petites expériences extra-sensorielles

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By Richard Wiseman, Pierre Bertrand 
| Dunod Editeur
Télépathie, voyance, hypnose... Le paranormal à l'épreuve de la science Voulez-vous contrôler vos rêves, flotter au-dessus de votre corps, dialoguer avec les morts ou convaincre de parfaits inconnus que vous savez tout d’eux ? Il est temps de vous révéler les secrets des phénomènes paranormaux !Richard...
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The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA damage

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By Georgios Efthymiou, Ziqi Song, Muddassir Malik, Eugénie Lohmann, Cláudio Montenegro, Paraskevi Kousteridou, Pierre Bertrand , Julia Pereira, Heather Carr, Issam Ben-Sahra, Coralie Compare, Ivan Nemazanyy, Pascal Finetti, François Bertucci, Christophe Lachaud, Ghislain Bidaut
| Science Magazine
Abstract The excessive production of extracellular matrix (ECM) and the metabolic adaptations in pancreatic ductal adenocarcinoma (PDAC) contribute individually to enhanced chemoresistance, marked tumor progression, and dismal patient survival. However, ECM-driven metabolic alterations that promote chemoresistance in PDAC are so far unexplored. Here, we use in vitro–generated ECM bioscaffolds that recapitulate cell-ECM interactions and induce broad metabolic alterations in PDAC cells.
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