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Ying-Qing Li on Muck Rack

Ying-Qing Li

As seen in: Nature, Journal of Biomedical Science, The Journal of Clinical Investigation
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Articles

LC3-dependent intercellular transfer of phosphorylated STAT1/2 elicits CXCL9+ macrophages and enhances radiation-induced antitumor immunity

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By Jun-Yan Li, Ying-Qing Li, Jia-Hao Dai, Qing-Mei He
| The Journal of Clinical Investigation
Abstract The efficacy of anticancer treatments, including radiotherapy, depends on the activation of type I IFN signaling. However, its regulatory networks and mechanisms remain to be elucidated. Here, we report that tumor cell–intrinsic type I IFN signaling can be transferred to macrophages via secretory autophagy, inducing CXCL9hi macrophages and enhancing CD8+ T cell–mediated antitumor immunity.
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PJA1-mediated suppression of pyroptosis as a driver of docetaxel resistance in nasopharyngeal carcinoma - Nature Communications

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By Yin Zhao, Jun-Yan Li, Ying-Qin Li, Sha Xu, Ying-Qing Li
| Nature Verified
Abstract Chemoresistance is a main reason for treatment failure in patients with nasopharyngeal carcinoma, but the exact regulatory mechanism underlying chemoresistance in nasopharyngeal carcinoma remains to be elucidated. Here, we identify PJA1 as a key E3 ubiquitin ligase involved in nasopharyngeal carcinoma chemoresistance that is highly expressed in nasopharyngeal carcinoma patients with nonresponse to docetaxel-cisplatin-5-fluorouracil induction chemotherapy.
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Differential genome-wide profiling of alternative polyadenylation sites in nasopharyngeal carcinoma by high-throughput sequencing

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By Na Liu, Ya-Fei Xu, Ying-Qing Li, Qing-Mei He
| Journal of Biomedical Science Verified
APA allows a single gene to encode multiple mRNA transcripts by changing the mRNA 3’UTR length and plays an important role in various physiological and pathological processes, including tumorigenesis [27, 30, 45, 46]. In this study, we performed genome-wide profiling of tandem APA sites in NPC and NNET tissues using SAPAS based on second-generation sequencing technology.
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