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HDAC5 depletion promotes hyper-acetylation of FOXA1 and potentiates HIF1α transcriptional activation in pancreatic cancer
Keywords pancreatic cancer HDAC5 FOXA1 HIF1α Get full text access Log in, subscribe or purchase for full access. References 1. Bray, F. ∙ Laversanne, M. ∙ Sung, H. ... Global cancer statistics 2022: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries CA Cancer J. Clin. 2024; 74:229-263 2. Del Chiaro, M. ∙ Sugawara, T. ∙ Karam, S.D. ... Advances in the management of pancreatic cancer Br. Med. J. (Clin. Res. Ed). 2023; 383, e073995 3. Lupien, M. ∙ Eeckhoute, J. ∙ Meyer, C.A. ...
Enhanced Lithium Storage Performance in Si/MXene Porous Composites
3.2. Electrochemical Characterization The electrochemical properties of the samples as negative electrode materials in LIBs were evaluated in half-cells. The electrochemical properties of NH2-Si1/MXene1, NH2-Si3/MXene1, NH2-Si5/MXene1, MXene, and pure Si were investigated. Figure 6a shows the CV test of the NH2-Si3/MXene1 composite in the potential interval of 0.01 to 1.5 V (vs. Li+/Li).
HDAC5 Loss Impairs RB Repression of Pro-Oncogenic Genes and Confers CDK4/6 Inhibitor Resistance in Cancer
Molecular Cell Biology Yingke Zhou, Xin Jin, Jian Ma, Donglin Ding, Zhenlin Huang, Haoyue Sheng, Yuqian Yan, Yunqian Pan, Ting Wei, Liguo Wang, Heshui Wu and Haojie Huang DOI: 10.1158/0008-5472.CAN-20-2828 Published March 2021 This article requires a subscription to view the full text. You may purchase access to this article or login to access your subscription using the links below.
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