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Yinglei Miao on Muck Rack

Yinglei Miao

As seen in: Nature, PLOS ONE, Frontiers in Immunology, Cell Host & Microbe
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Articles

RNF213 marks a regulatory T-cell subset associated with ulcerative colitis

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By Jing Wu, Jiayi Yang, Lianyu Zhou, Lifang Chen, Yunxing Li, Ruijie Ma, Yinglei Miao, Jie Jia, Jiarong Miao, Zhangqin Li, Youxu Ren
| Frontiers in Immunology
Abstract Background: Ulcerative colitis (UC) is a nonspecific autoimmune disease, the etiology and pathogenesis of which remain unclear. Identifying new disease targets could inform clinical diagnosis and treatment. Methods: We integrated single-cell RNA sequencing (scRNA-seq) and bulk transcriptome data from UC patients. We screened key biomarkers and constructed a nomogram diagnostic model through three machine learning algorithms (LASSO, SVM-RFE, and Boruta).
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Identification of antimicrobial peptides from ancient gut microbiomes - Nature Communications

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By Sizhe Chen, Ye Peng, Hein Min Tun, Zhimin Jiang, Yinglei Miao, Sunjae Lee, Xiaole Yin, Yang Sun, Siew Chien Ng
| Nature Verified
Abstract Fecal coprolites preserve ancient microbiomes and are a potential source of extinct but highly efficacious antimicrobial peptides (AMPs). Here, we develop AMPLiT (AMP Lightweight Identification Tool), an efficient tool deployable to portable hardware for AMP screening in metagenomic datasets. AMPLiT demonstrates AUPRC performances of 0.9486 ± 0.0003 and reasonable overall training time of 3200 ± 53 s.
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Heat shock transcription factor 2 reduces mitochondrial pathway apoptosis in intestinal epithelial cells by inhibiting the increase in mitochondrial membrane permeability in ulcerative colitis

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By Wen Wang, Juan Luo, Yinglei Miao, Yunling Wen
| PLOS ONE
Citation: Wang W, Wen Y, Luo J, Miao Y, Zhang F, Niu J (2025) Heat shock transcription factor 2 reduces mitochondrial pathway apoptosis in intestinal epithelial cells by inhibiting the increase in mitochondrial membrane permeability in ulcerative colitis. PLoS One 20(5): e0325275.
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