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C5aR1+ microglia exacerbate neuroinflammation and cerebral edema in acute brain injury
Keywords cerebral edema microglial heterogeneity C5aR1+ microglia neurotoxic astrocytes neutrophils recruitment Get full text access Log in, subscribe or purchase for full access. References 1. Jiang, J.Y. ∙ Gao, G.Y. ∙ Feng, J.F. ... Traumatic brain injury in China Lancet Neurol. 2019; 18:286-295 2. Urday, S. ∙ Kimberly, W.T. ∙ Beslow, L.A. ... Targeting secondary injury in intracerebral haemorrhage--perihaematomal oedema Nat. Rev. Neurol. 2015; 11:111-122 3. McCrea, M.A. ∙ Giacino, J.T. ∙ Barber, J. ...
20‐HETE synthesis inhibition attenuates traumatic brain injury-induced mitochondrial dysfunction and neuronal apoptosis via the SIRT1/PGC‐1α pathway: A translational study - Cui - - Cell Proliferation - Wiley Online Library
1 INTRODUCTION Traumatic brain injury (TBI) is a leading cause of death and disability worldwide. Alleviation of secondary brain injury and promotion of neurological recovery after TBI are key to reducing the burden on affected families and society and to improving the quality of life of patients.1, 2 TBI involves two pathological processes: primary brain injury and secondary brain injury.
Diversity and space-time dynamics of the bacterial communities in cotton (Gossypium hirsutum) rhizosphere soil
YingWu Shi,ab HongMei Yang,ab Ming Chu,ab XinXiang Niu,c XiangDong Huo,ab Yan Gao,ab Jun Zeng,ab Qing Lin,ab Tao Zhang,ab YuGuo Li,ab KuEr Outi,ab Kai Lou,ab XueYan Li,ab WenFang Dang,ab Tao Zhangab aInstitute of Microbiology, Xinjiang Academy of Agricultural Sciences, Urumqi 830091, Xinjiang, P.R. China. bXinjiang Laboratory of Special Environmental Microbiology, Urumqi 830091, Xinjiang, P.R. China.
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