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Yingyu Huang

As seen in: Wiley, Wiley Online Library, ScienceDirect, MDPI, ZDNet, ACS Publications, Royal Society of Chemistry, Cell Press, JoVE- Journal of Visualized Experiments
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Articles

Unbalanced chromatin binding of Polycomb complexes drives neurodevelopmental disorders

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By Rodrigo Borges, Gretter González-Blanco, Harikumar Arigela, Yingyu Huang, Lucas Caeiro, Nikolai Fattakhov, Stefano Lepore, Liliana Garcia-Martinez, Matea Maurice, Pushti D. Mehta, Emily Park, Kailynn MacGillivray, Jevithen Nehru, Matthew Chau, Maria C. Robayo, Clemer Abad, Alicia Bilbao-Martinez, Fabiola Monteiro, XI LUO, Daniel Bilbao, Simone Sidoli, Bruno Di Stefano, Katherina Walz, Arneet L. Saltzman, Ramiro E. Verdun, Ramin Shiekhattar, Lluis Morey
| Cell Press Verified
STAR★Methods REAGENT or RESOURCESOURCEIDENTIFIER Antibodies H3K27me3 (ChIP-seq, CUT&RUN, WB) Active Motif Cat# 39155, RRID:AB_2461020 H2AK119ub (ChIP-seq, CUT&RUN, WB) Thermo Fisher Scientific Cat# 720148, RRID:AB_2605927 Ring1b (ChIP-seq) Cell Signaling Technology Cat# 5694, RRID:AB_10705604 CBX7 (ChIP-seq) Abcam Cat# ab21873, RRID:AB_726005 RYBP (ChIP-seq, WB) EMD Millipore Cat# AB3637, RRID:AB_2285466 MTF2 Polyclonal antibody (ChIP-seq) PROTEINTECH Cat# 16208-1-AP, RRID:AB_2147370 JARID2...
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pH-Responsive Self-Assembly of Renal-Clearable Nanoparticles in the Kidneys: One Assembly per Endosome

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By Yingyu Huang, Jie Zheng, Mengxiao Yu
| ACS Publications
Cite Jump to ExpandCollapse ArticleSeptember 12, 2025 pH-Responsive Self-Assembly of Renal-Clearable Nanoparticles in the Kidneys: One Assembly per Endosome Click to copy article linkArticle link copied! Yingyu Huang Yingyu Huang Department of Chemistry and Biochemistry, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, Texas 75080, United States Jie Zheng* Jie Zheng Department of Chemistry and Biochemistry, The University of Texas at Dallas, 800 W.
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Mechano-oncogenic cytoskeletal remodeling drives leukemic transformation with mitochondrial vesicle-mediated STING activation

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By Yingyu Huang, Yu ZHANG, Manhui Wei, Xinglong Liu
| ScienceDirect Verified
Acute myeloid leukemia (AML) is the most common and aggressive form of adult leukemia, arising from mutated hematopoietic stem cells and myeloid progenitors (MPs) and frequently driven by chromosomal translocations as early genetic events.1,2,3 Despite advancements in treatment, over 70% of AML patients succumb to the disease within 5 years, primarily due to drug resistance and relapse.4 Understanding the molecular mechanisms underlying leukemogenesis is, therefore, critical for identifying...
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