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Hans-Joachim Anders

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Freiburg
As seen in: Nephrology Dialysis Transplantation, Nature, Oxford University Press (OUP), Annals of the Rheumatic Diseases, MDPI, Journal of Biomedical Science, Science Magazine, The New England Journal of Medicine, Cell Press, Canadian Journal of Infectious Diseases & Medical Microbiology, Journal of the American Society of Nephrology (JASN) and
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Articles

CCR5 and CD74 are potential therapeutic targets for necroinflammation in preclinical cholesterol crystal embolism

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By Chongxu Shi, Matthieu Galtier, Adam S. Sperling, Zhaozhi Wen, Koulong Zheng, Bowen Li, Huiping Miao, Barbara M. Klinkhammer, Peter Boor, Hans-Joachim Anders Verified, Dong Liu
| Science Magazine Verified
Editor’s summary Abstract Supplementary Materials REFERENCES AND NOTES Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Editor’s summary Cholesterol crystal embolism (CCE) is a complication of atherosclerosis that is potentially life-threatening. Emboli can result in ischemic tissue damage including acute kidney injury (AKI) for which there are limited therapeutic options. Shi et al.
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Author Correction: Cleavage of roquin and regnase-1 by the paracaspase MALT1 releases their cooperatively repressed targets to promote TH17 differentiation | Nature Immunology

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By Desheng Hu, Sven Brenner, Jessica Zöller, Gitta A. Heinz, Daniel Nagel, Katharina U. Vogel, Nina Rehage, Renee Gloury, Nina M Martin, Maciej Lech, Arie Geerlof, Achim Weber, Hans-Joachim Anders Verified, Ingo Schmitz, Marc Schmidt-Supprian, Mingui Fu, Helmut Holtmann, Daniel Krappmann, Jürgen Ruland, Axel Kallies, Mathias Heikenwalder, Vigo Heissmeyer, Katharina M. Jeltsch, Sebastian C. Warth, Stephanie L. Edelmann, Claudia Lohs, Jenny E. Stehklein, Elisabeth Kremmer
| Nature Verified
Correction to: Nature Immunology https://doi.org/10.1038/ni.3008, published online 5 October 2014. In the version of this article initially published, due to a copy / paste error during figure preparation, in Fig. 7f, the WT plot was an inadvertent duplicate of the adjacent R510G,R579G plot. Due to the age of the article, the figure cannot be updated directly. The corrected Fig. 7f is available as Supplementary Information accompanying this amendment. This notice serves to correct the article.
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Multifactorial chronic kidney disease and the kidney capacity–workload balance - Nature Reviews Nephrology

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By Paola Romagnani, Hans-Joachim Anders Verified
| Nature Verified
Abstract Chronic kidney disease (CKD) is defined by the presence of kidney abnormalities with an impact on health that lasts more than 3 months. Cross-sectional population data suggest that ~10% of the world population might be affected. CKD is often classified by aetiology, under the assumption that a single cause drives disease, and identifying the single cause that drives kidney disease in a patient is a key nephrology expertise taught at medical schools.
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