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Marie Johnson on Muck Rack

Marie Johnson

As seen in: PLOS, Substack, BioRxiv, Cell Reports, betanews, Ladders, East Bay Express, AZ Big Media, Martech Series, CPO Magazine and
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Articles

MYCN drives pediatric glioma transformation from neural progenitors and creates distinct therapeutic vulnerabilities

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By Taylor A. Gatesman, Srinidhi Varadharajan, Brenden Johnson, Ryan J. Duchatel, Rakesh Pathak, Andrea Cruz, Hannah Wolfe, Marie Johnson, Fengping Li, Akanksha Chilukuri, Matthew E. Halbert, Tritan J. Plute, Jennifer Perez, Jennifer Pérez, Joshua Michel, Zachary C. Gersey, Sruti Shiva, Gary Kohanbash, Ales Vicha, Michal Zapotocky, James Felker, Shaun Carlson, thomas pearce, George K. Gittes, Baoli Hu, Edward V. Prochownik, Xiuxing Wang, Yijen Wu, Taylor J. Abel, Ian F. Pollack, Jeremy Rich, Matthew Dun, Stephen Mack, Sameer Agnihotri, Katharine Halligan, Lily G. Garton, Karensa Tjoa, Daniel R. David Premkumar, Megan Mantica
| Cell Reports Verified
Keywords MYCN amplification pediatric high grade glioma neural progenitor cells PI3K mTOR signaling Paxalisib insulin IGF feedback ketogenic diet Research topic(s) CP: cancer Introduction Pediatric high-grade gliomas (pHGGs) are a rare cancer with an approximate incidence of 0.85 per 100,000, accounting for 15%–20% of all pediatric central nervous system (CNS) tumors.1,2 pHGGs are a histologically and molecularly heterogeneous group of tumors, varying from anaplastic astrocytoma (WHO grade...
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Beyond Logistics: Applying Neuroscience Strategies to Your Meetings & Events Portfolio | M&IW

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By Marie Johnson
| Events Forward
Back to News & Info Organizations invest millions in events each year, including sales kickoffs, product launches, leadership summits, incentive travel, and user conferences. These events aren’t just about logistics; they’re strategic levers for growth. But stakeholders struggle to measure value and outcomes across a portfolio of events, making it difficult to demonstrate ROI, optimize budgets, and align initiatives with strategic business goals.
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Targeting Galectin-3 to modulate inflammation in LAMA2-deficient congenital muscular dystrophy

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By Jinseo Lee, Marie Johnson, Jia Qi Cheng-Zhang, Ruvindi N Ranatunga
| BioRxiv
Abstract LAMA2-deficient congenital muscular dystrophy (LAMA2-CMD) is a severe neuromuscular disorder characterized by muscle degeneration, chronic inflammation, and fibrosis. While inflammation is one the hallmarks of LAMA2-CMD, the immune cell composition in laminin-deficient muscles remains understudied. Consequently, targeted pharmacological intervention to reduce inflammation remains underexplored.
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