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Prachi Gupta

As seen in: Amazon Web Services, Medium, MSN, MSN Canada, Yahoo Life, HuffPost, Nature, Scribd, The Guardian, The Washington Post and
barely on here. find me on IG @prachigu or email prachigu at protonmail | THEY CALLED US EXCEPTIONAL out now

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Prachi Gupta’s Journalist Portfolio

Enterprise Mobility Strategy - Designing to Succeed

Enterprise Mobility Strategy - Designing to Succeed

Nothing lasts in the IT industry except its lessons. And if there is one lesson that has been resoundingly cl…

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App Monetization Strategy - How To Make Money Through In-App Advertising

App Monetization Strategy - How To Make Money Through In-App Advertising

Most people in the tech world remember 2007 as the year mobility had changed forever. Steve Jobs took stage a…

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Articles

Linker Chemistry in Antibody–Drug Conjugates: Current Strategies, Clinical Pharmacokinetic Analyses, and Linker-Free Outlooks Click to copy article link Article link copied!

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By Theeraphop Prachyathipsakul, Prachi Gupta, Seth K. Thayumanavan
| ACS Publications
Cite Jump to ExpandCollapse ViewpointJuly 23, 2026 Linker Chemistry in Antibody–Drug Conjugates: Current Strategies, Clinical Pharmacokinetic Analyses, and Linker-Free Outlooks Click to copy article linkArticle link copied! Theeraphop Prachyathipsakul Theeraphop Prachyathipsakul Department of Chemistry, University of Massachusetts, Amherst, Massachusetts 01003, United States Prachi Gupta Prachi Gupta Department of Chemistry, University of Massachusetts, Amherst, Massachusetts 01003, United...
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Building agentic AI applications with a modern data mesh strategy on AWS

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By Venkata Sistla, Aamna Najmi, Prachi Gupta
| Amazon Web Services
When a customer service agent autonomously queries order databases, retrieves return policies, and synthesizes answers, it needs governed access to multiple data sources across your organization. Building agentic AI applications on a modern data mesh requires fine-grained access control enforced at every layer of the data interaction chain.
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Rapid activation of ARF6 after RAF inhibition augments BRAF V600E and promotes therapy resistance

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By Junhua Wang, Yinshen Wee, Thomas Jacob, Aaron Rogers, Deja M. Brooks, Prachi Gupta, Emily Wilson Verified, tong liu, Eric Smith, michael davies, Martin McMahon, martin mcmahon, Sheri Holmen, Robert L. Judson-Torres, Roger Wolff, Allie H. Grossmann, Lise. K. Sorensen, Joshua K. H. Tay, YN Vashisht Gopal
| Nature Verified
Abstract The intrinsic ability of cancer cells to evade death underpins tumorigenesis, progression, metastasis, and the survival of drug-tolerant persister (DTP) cells. Herein, we discovered that the small GTPase ARF6 plays a central role in tumor survival by fortifying RAF oncoprotein levels. ARF6 activation was sufficient to increase BRAFV600E, ARAF, and CRAF proteins through a post-transcriptional mechanism, while sustained inhibition of ARF6 eventually led to decay.
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