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Tieling Zhou

As seen in: Science Magazine, Cell Press
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Articles

Spatial multi-omics landscape of colorectal cancer macro- and micrometastases

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By Yang Liu, Akshaya Jadhav, Yuwen Pan, Jianlong Liao, Isha Khanduri, Yunhe Liu, Riham Katkhuda, Wei Lu, Kyung Serk Cho, Zhimin Tong, Tieling Zhou, Kangyu Lin, Baohua Sun, Mei Jiang, Sharia Hernandez, Patrick Brennan, Guangsheng Pei, Kai Yu, Yibo Dai, Tian Ren Chu, Fuduan Peng, Ling Xia, Youming Guo, Alicia Mejia, Sean Barnes, Ou Shi, Alaa Mohamed, Natalie Fowlkes, Timothy Newhook, Yun Shin Chun, Van Morris, David G. Menter, Dadi Jiang, John Paul Shen, Ryan Sun, Jean-Nicolas Vauthey, Ruoyan Li, Humam Kadara, Scott Kopetz, Linghua Wang, Dipen M. Maru, Saxon Rodriguez, Shreeya Indulkar, Luisa M. Solis Soto
| Cell Press Verified
Introduction While early detection through screening has significantly reduced the incidence of localized colorectal cancer (CRC),1 limited understanding of the biological mechanisms driving colorectal metastasis remains a major barrier to improving long-term survival.
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Pan-cancer spatial atlas of tertiary lymphoid structures

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By Oliver Lindecke, Maria Bue, Marion Clavel, Kyung Serk Cho, Yunhe Liu, Guangsheng Pei, Jianfeng Chen, Yibo Dai, Yang Liu, Tieling Zhou, Antoine Bougoüin, Alejandra Serrano, Khalida Wani, Akshaya Jadhav, Jimin Min, Sharia Hernandez, Wei Lu, Daiwei Zhang, Jiahui Jiang, Diana Shamsutdinova, Enyu Dai, Fuduan Peng, Ansam Sinjab, Paola A. Guerrero, Kai Yu
| Science Magazine Verified
Editor’s summary Structured Abstract Abstract Supplementary Materials References and Notes Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Editor’s summary Tertiary lymphoid structures (TLSs) form local immune hubs inside tumors, but they are diverse and not all are equally functional. Cho et al. built a pan-cancer atlas and developed an artificial intelligence (AI)–based framework to detect and characterize TLSs in human tumors.
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Multimodal spatial-omics reveal co-evolution of alveolar progenitors and proinflammatory niches in progression of lung precursor lesions

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By Fuduan Peng, Ansam Sinjab, Yibo Dai, Warapen Treekitkarnmongkol, Sujuan Yang, Tieling Zhou, Minyue Chen, Alejandra Serrano, Avantika B Krishna, Nastaran Karimi, Manvi Sharma, Akshay V Basi, Guangsheng Pei, Jianlong Liao, Yunhe Liu, Jiping Feng, Zahraa Rahal, Yang Liu, Jiahui Jiang, Kai Yu, Yuejiang Liu, Kyung Serk Cho, Jichao Chen, Luisa M. Solis, Sarah A. Mazzilli, Steven M. Dubinett, Tina Cascone, Avrum Spira, Stephen Swisher, Naoe Jimbo, Takuo Hayashi, Satsuki Kishikawa, Kazuya Takamochi, Tomoo Itoh, Takashi Yao, Kenji Suzuki, Neda Kalhor, Ignacio I. Wistuba, Mingyao Li, Seyed Javad Moghaddam, Junya Fujimoto, Jared K. Burks, Jeffrey Myers, Kadir C. Akdemir, Linghua Wang, Humam Kadara
| Cell Press Verified
Keywords lung adenocarcinoma lung precursor lesions spatial transcriptomics Xenium in situ alveolar progenitors reactive type II pneumocytes proinflammatory pathways IL-1β epithelial-immune niche LUAD interception Introduction Lung adenocarcinoma (LUAD) is increasingly being diagnosed in smokers and never-smokers and has become the dominant histological subtype of lung cancer.1 Despite clinical advances, mortality of patients with advanced LUAD remains relatively high.2 Improving outcomes for...
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