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Conflicts of Interest The authors declare no conflicts of interest. Data Availability Statement The data that supports the findings of this study are available in the supplementary material of this article. Supporting Information Filename Description adfm78544-sup-0001-SuppMat.docx34.9 MB Supporting File 1: adfm78544-sup-0001-SuppMat.docx. adfm78544-sup-0002-SuppMat.docx34.9 MB Supporting File 2: adfm78544-sup-0002-SuppMat.docx.
ArticleOnline nowOpen access Affiliations & Notes 1Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, MA, 02215, United States of America 2Department of Genetics, Blavatnik Institute, Harvard Medical School, Boston, MA, 02115, United States of America 3Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, MA, 02215, United States of America 4Department of Immunology, Harvard Medical School, Boston, MA, 02115, United States of America 5Systems,...
Editor’s summary Structured Abstract Abstract Supplementary Materials References and Notes Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Editor’s summary Dendritic cells (DCs) are essential for cancer immunity because they prime tumor-specific T cells. Therefore, a pharmacological strategy to selectively enhance DC function could be desirable as a cancer immunotherapy. Zhang et al.
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