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Abstract Decades of structural magnetic resonance imaging (MRI) studies have documented alterations of gray matter morphometry in psychiatric disorders, but the field has failed to identify any consensus disease phenotypes.
Abstract The cerebral cortex is organized along a dominant sensorimotor-to-association (S–A) axis, anchored by modality-specific primary sensorimotor areas at one end and transmodal association areas forming distributed networks that support abstract cognition at the other1,2,3,4,5,6,7,8,9,10,11. The developmental mechanisms shaping this axis remain unclear9,10,11,12,13,14,15,16,17,18,19,20,21,22,23,24.
Abstract Converging neuroimaging, genetic, and post-mortem evidence highlights the fundamental role of synaptic density reductions in schizophrenia pathogenesis. However, the brain-wide spatial pattern of these alterations and the mechanisms underlying this patterning remain to be established.
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