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Abstract Sex differences in the prevalence, clinical phenotypes and therapeutic responses of rheumatic diseases have been recognized for decades, but the underlying mechanisms remain largely unknown. Accumulating evidence highlights the critical roles of both immune and non-immune cells in disease pathogenesis and the influence of sex hormones on cellular function.
Abstract The cerebral cortex is organized along a dominant sensorimotor-to-association (S–A) axis, anchored by modality-specific primary sensorimotor areas at one end and transmodal association areas forming distributed networks that support abstract cognition at the other1,2,3,4,5,6,7,8,9,10,11. The developmental mechanisms shaping this axis remain unclear9,10,11,12,13,14,15,16,17,18,19,20,21,22,23,24.
Supporting Information Filename Description art70163-sup-0001-Supinfo.zipZip archive, 11.8 MB Data S1: Supporting Information art70163-sup-0002-Disclosureform.pdfPDF document, 671 KB Data S2: Disclosureform
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