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Xiaotu Ma

As seen in: Nature, Genome Biology, Genome Medicine, Phys.org, Royal Society of Chemistry, BioRxiv, Cell Press, Cell Reports
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Articles

Author Correction: Sustained nitric oxide production by engineered E. coli remodels the tumor microenvironment and potentiates immunotherapy

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By Shuyu Xu, Tianjiao Zhang, Mengxin Wang, Ruiqi Wu, Mofan Li, Haonan Wang, Xinxin Xie, Qingfeng Chen, Xiaotu Ma, Xiaolong Liang
| Nature Verified
Correction to: Nature Biotechnology https://doi.org/10.1038/s41587-026-03054-y, published online 18 March 2026. In the version of this article initially published, there was an error in the Fig. 4n Source Data, where an additional “anti-EGFR” dataset was accidentally included; an error in the Fig. 6d Source Data, with errors in the PBS group measurements, also affecting the plots in the article; errors in the Figs. 4o, 5b–d, h Source Data, due to data entry errors; and errors in the Fig.
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Sustained nitric oxide production by engineered E. coli remodels the tumor microenvironment and potentiates immunotherapy

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By Shuyu Xu, Tianjiao Zhang, Mengxin Wang, Ruiqi Wu, Mofan Li, Haonan Wang, Xinxin Xie, Qingfeng Chen, Xiaotu Ma, Xiaolong Liang
| Nature Verified
Abstract Tumor immunotherapy is often compromised by an immunosuppressive tumor microenvironment (TME) characterized by abnormal vasculature and exhausted T cells. Here, given the role of nitric oxide (NO) in favorably remodeling the TME, we engineered Escherichia coli Nissle 1917 (ECN) with a synthetic arginine–NO circuit (ECN-NO) that modifies the arginine synthesis pathway to constitutively synthesize arginine and enable sustained NO production.
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Developmental reprogramming underlies chemotherapy resistance in favorable-histology Wilms tumor

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By Andrew Fleming, Carolyn M Jablonowski, Hongjian Jin, Siwei Zhang, Surbhi Sona, Karissa Jones, Changde Cheng, Beisi Xu, Christopher Morton, Mary A. Woolard, Prahalathan Pichavaram, Daniel B. Gehle, Sivaraman Natarajan, Kiran Kodali, Vishwajeeth Pagala, Anthony High, Yogesh Kumar, Steven J. Burden, Virginia Valentine, Jake Harbour, Daniel Vocelle, Ti-Cheng Chang, John Easton Verified, Scott Olsen, Geoffrey Neale, Emilia M. Pinto, Jerold E. Rehg, Laura J. Janke, Teresa Santiago, Rani E. George, Xiaotu Ma, Gerard P. Zambetti, Andrew Davidoff, Taosheng Chen, Gang Wu, Xiang Chen, Jun Yang, Andrew Murphy, Deidre Daria
| Cell Reports Verified
Keywords Wilms tumor chemotherapy resistance LIN28B ABCB1 N-MYC MYCN kidney development Research topic(s) CP: cancer Introduction Wilms tumor (WT) is the most common pediatric renal malignancy.1 WT is an embryonal tumor with triphasic, mixed histology consisting of blastemal, epithelial, and stromal elements that mirror cell types in the developing kidney.
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