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Ying Xing

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Articles

Myeloid-derived suppressor cells in cancer: biology, regulatory networks and theranostic prospects

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By Jue Yang, Ying Xing, Yuxing Yao, Xiaojuan He, Aiping Lyu, Yuanyan Liu, Rongjun Deng, Xiuyun Bai, Qiqiong Liu
| Nature Verified
Abstract Despite the remarkable progress in cancer treatment, drug resistance and immune escape still severely limit clinical efficacy, largely due to tumor-induced immunosuppression. The main driver of this suppressive environment is myeloid-derived suppressor cells (MDSCs).
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Tirofiban for Branch Atheromatous Disease–Related Stroke : The STRATEGY Randomized Clinical Trial

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By Yicong Wang, Xiaoling Liao, Shuo Feng, Yuesong Pan, Xuan Wang, Hui Qu, YueTong Liu, Cong Gao, Xinru Liu, Qianqian Yang, Meiyang Zhang, Zhangxinyi Liu, Jinyi Ye, Yuke Mao, Weiqi Chen, Lingling Jiang, Philip M. Bath, S. Claiborne Johnston, Guillaume Turc, Xingquan Zhao, Yongjun Wang, Yilong Wang, Hongliang Wang, Caijun Zhao, Haijun Qiu, Hongqin Yang, Yingzhuo Zang, xinqiang Wang, Wei Zhang, Jinguo Zhao, Lili Zhao, Shuo Zhang, Yanan Zheng, Hao Yue Zhu, Ying Li, Yuanwei Wang, Jianhua Li, JingXian Fang, Xiting Zhang, Dongqi Liu, Huqing Li, Zengqiang Sun, Ping Shen, Qingfang Xie, Ying Xing, Xinhui Li, Xiao Hu, Jing Liu, Fuqiang Wang, Yifeng Du, Aijun Ma, Lina Xu, Guangming Kang, Panbing Huang, Wusheng Lu, Wen Shangguan, Yuanliang Cui, Chengguang Song
| JAMA Network Verified
Tirofiban for Branch Atheromatous Disease–Related Stroke: The STRATEGY Randomized Clinical Trial Supplement 1. Supplement 2. Statistical analysis plan Supplement 3. eAppendix 1. Listing of committees in STRATEGY Trial eAppendix 2. Participating sites and investigators in STRATEGY Trial eAppendix 3. Inclusion and exclusion criteria eAppendix 4. Imaging details eAppendix 5. Study design and treatment allocation eAppendix 6. Definition of outcomes eFigure 1.
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p53 hotspot mutants attenuate CTL-mediated tumor cell killing through a novel ALKBH5-YTHDF3-PD-L1 pathway

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By Zhuan Ju, Xiang Zhang, Wenjie Zuo, Lu Zhang, Xin Fan, Chenxing Zhang, Xiaofang Zhang, Xiao Jun Zhang, Yidi WANG, Ying Xing, Baile Zuo, Angang Yang, Rui Zhang, Huilong Yin
| Nature Verified
Abstract TP53 is a well-documented tumor suppressor gene frequently mutated in malignancies. It has been demonstrated that the gain-of-function (GOF) mutation of p53 promotes the development and progression of cancers; however, its extrinsic oncogenic mechanisms are still poorly understood. Herein, we found that the oncogenic effect of mutant p53 in vivo is dependent on the tumor immune microenvironment.
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